C
Charles E. Alpers
Researcher at University of Washington
Publications - 418
Citations - 35650
Charles E. Alpers is an academic researcher from University of Washington. The author has contributed to research in topics: Renal pathology & Glomerulonephritis. The author has an hindex of 89, co-authored 409 publications receiving 32440 citations. Previous affiliations of Charles E. Alpers include New York Medical College & University of Washington Medical Center.
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Focal segmental glomerulosclerosis in primates infected with a simian immunodeficiency virus
Charles E. Alpers,Che-Chung Tsai,Kelly L. Hudkins,Yan Cui,LaRene Kuller,Raoul E. Benveniste,Jerrold M. Ward,William R. Morton +7 more
TL;DR: Autopsy studies indicate SIV-infected primates may provide a relevant model for study of human HIV-associated nephropathy and indicate the variable pathology that can be seen in primate infections of distinct retrovirus types, each of which produces a simian immunodeficiency state that resembles human AIDS.
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AJKD Atlas of Renal Pathology: Thrombotic Microangiopathy
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AJKD Atlas of Renal Pathology: diabetic nephropathy.
TL;DR: In type 1 diabetes, interstitial fibrosis and tubular atrophy follow glomerular lesions and may be less severe or proportional to diabetic glomerulopathy, and in type 2 diabetes, in which arteriosclerosis is commonly present, the lesions are more heterogeneous, and chronic tubulointerstitial injury may be more severe than the diabetic glomersulopathy.
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Monoclonal Immunoglobulin Deposition Disease in a Renal Allograft: Probable Recurrent Disease in a Patient Without Myeloma
TL;DR: A case in which monoclonal IgG-kappa deposition in the glomeruli of a renal allograft apparently resulted in graft failure is described, indicating the potential for disease recurrence even in the absence of a systemic lymphoplasmacytic disorder.
Journal Article
Mechanisms of clearance of immune complexes from peritubular capillaries in the rat
TL;DR: Efficient clearance of IC in PC compared with GC may be due to differences in hemodynamic forces, amounts of IC formed in each of these sites, differences in binding of IC to subendothelial basement membrane, or phenotypic specialization of the endothelium lining these two different capillary beds.