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Charles E. Alpers

Researcher at University of Washington

Publications -  418
Citations -  35650

Charles E. Alpers is an academic researcher from University of Washington. The author has contributed to research in topics: Renal pathology & Glomerulonephritis. The author has an hindex of 89, co-authored 409 publications receiving 32440 citations. Previous affiliations of Charles E. Alpers include New York Medical College & University of Washington Medical Center.

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Journal ArticleDOI

Interstitial eosinophilic aggregates in diabetic nephropathy: allergy or not?

Dao-Fu Dai, +6 more
- 01 Aug 2015 - 
TL;DR: In DN, IEA are associated with the severity of IFTA but not with prescribed medications or clinical history of allergy, which suggests that in DN I EA are often associated with chronic tubulointerstitial injury and are not diagnostic of an allergic interstitial nephritis.
Journal Article

Effects of aging on expression of ischemic acute renal failure in rats.

Zager Ra, +1 more
- 01 Sep 1989 - 
TL;DR: It is concluded that aging increases susceptibility to severe ischemic acute renal failure in the rat, an effect that is apparent even during a transition from the adolescent to the mature state.
Journal ArticleDOI

Renal expression and activity of the germinal center kinase SK2

Andrey V. Cybulsky, +7 more
- 01 Jan 2004 - 
TL;DR: SK2 is a renal epithelial protein kinase whose expression and activity are increased during development and recovery from acute renal failure, where tubular epithelial regeneration may recapitulate developmental processes.
Journal ArticleDOI

Platelet-Derived Growth Factor–A mRNA Expression in Fetal, Normal Adult, and Atherosclerotic Human Aortas Analysis by Competitive Polymerase Chain Reaction

Charles E. Murry, +4 more
- 15 Mar 1996 - 
TL;DR: The significance of high levels of PDGF-A mRNA in the "quiescent" aortic media is unknown but it clearly does not promote cell replication.
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Bone morphogenetic protein 4: potential regulator of shear stress-induced graft neointimal atrophy.

Patrick C.H. Hsieh, +12 more
- 01 Jan 2006 - 
TL;DR: Increased BMPs (particularly BMP4) coupled with decreased noggin may promote high shear stress-mediated graft neointimal atrophy by inhibiting SMC proliferation and increasing SMC death.