C
Cynthia A. Tse
Researcher at University of Auckland
Publications - 10
Citations - 582
Cynthia A. Tse is an academic researcher from University of Auckland. The author has contributed to research in topics: Cell culture & Insulin resistance. The author has an hindex of 8, co-authored 10 publications receiving 565 citations.
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Journal ArticleDOI
Testosterone selectively reduces the high molecular weight form of adiponectin by inhibiting its secretion from adipocytes
Aimin Xu,Aimin Xu,Kok Weng Chan,Ruby L. C. Hoo,Yu Wang,Yu Wang,Kathryn C.B. Tan,Jialiang Zhang,Baoying Chen,Michael C. Lam,Cynthia A. Tse,Garth J. S. Cooper,Karen S.L. Lam +12 more
TL;DR: The selective inhibition of HMW adip onectin by testosterone might contribute to the sex dimorphism of adiponectin in terms of its oligomeric complex distribution and could partly explain why men have higher risk to insulin resistance and atherosclerosis than women.
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Alteration in Phosphorylation of P20 Is Associated With Insulin Resistance
TL;DR: In rats made insulin resistant by dexamethasone treatment, levels of the phosphoisoforms S2 and S3, which were barely detectable in healthy rats in the absence of hormone stimulation, were significantly increased and the ability of insulin to inhibit amylin-evoked phosphorylation of these two isoforms was greatly attenuated.
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Activation of activating transcription factor 2 by p38 MAP kinase during apoptosis induced by human amylin in cultured pancreatic β‐cells
TL;DR: It is shown that p38 kinase became activated by hA treatment of cultured β‐cells whereas extracellular signal‐regulated kinase (ERK) did not; by contrast, nonfibrillogenic rat amylin (rA) altered neither.
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Modulation of proliferation of cultured human cells by urinary trypsin inhibitor.
TL;DR: The trypsin inhibitor from human urine (UTI) is purified and a biphasic effect of this protein on the proliferation of human fibroblasts is demonstrated, and two types of cellular binding site which may be responsible for the stimulatory and inhibitory aspects of this effect are identified.
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Proteomic analysis of adipocyte differentiation: Evidence that α2 macroglobulin is involved in the adipose conversion of 3T3 L1 preadipocytes
TL;DR: The results suggest that hormone treatment induces differentiation at least in part by suppression of intracellular α2 macroglobulin activity in 3T3 L1 preadipocytes.