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Daniel Fourmy

Researcher at University of Toulouse

Publications -  103
Citations -  2369

Daniel Fourmy is an academic researcher from University of Toulouse. The author has contributed to research in topics: Receptor & Cholecystokinin. The author has an hindex of 30, co-authored 101 publications receiving 2180 citations. Previous affiliations of Daniel Fourmy include Paul Sabatier University & French Institute of Health and Medical Research.

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Targeting a G-protein-coupled receptor overexpressed in endocrine tumors by magnetic nanoparticles to induce cell death.

TL;DR: A nanoplatform composed of an iron oxide nanocrystal decorated with a ligand of a G-protein coupled receptor, the cholecystokinin-2 receptor that is overexpressed in several malignant cancers is designed, demonstrating that cell death is triggered even though nanoparticles of low thermal power are internalized in minute amounts by the cells.
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Targeted Magnetic Intra-Lysosomal Hyperthermia produces lysosomal reactive oxygen species and causes Caspase-1 dependent cell death.

TL;DR: Using Gastrin‐grafted MNPs specifically delivered to lysosomes of tumor cells from different cancers, evidence is provided that MILH causes cell death through a non‐apoptotic signaling pathway and highlights the clear potential of MILH for the eradication of tumors overexpressing receptors.
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Biologically active recombinant human progastrin(6-80) contains a tightly bound calcium ion.

TL;DR: It is concluded that recombinant human progastrin6–80 is biologically active and contains a single calcium ion, the first report of selective, high affinity binding of metal ions to a prohormone.
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Identification of determinants of glucose-dependent insulinotropic polypeptide receptor that interact with N-terminal biologically active region of the natural ligand

TL;DR: Identification of residues in the GIPR involved in functional interaction with N-terminal moiety of GIP represents an important step toward understanding activation of G IPR by GIP, which should facilitate the rational design of therapeutic agents.