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Deeann Wallis

Researcher at University of Alabama at Birmingham

Publications -  44
Citations -  2937

Deeann Wallis is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Neurofibromin 1 & Attention deficit hyperactivity disorder. The author has an hindex of 22, co-authored 41 publications receiving 2654 citations. Previous affiliations of Deeann Wallis include Texas A&M Health Science Center & Texas A&M University.

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Mutations in the homeodomain of the human SIX3 gene cause holoprosencephaly.

TL;DR: It is proposed that SIX3 is the HPE2 gene, essential for the development of the anterior neural plate and eye in humans, and Mutational analysis has identified four different mutations in the homeodomain of Six3 that are predicted to interfere with transcriptional activation and are associated with HPE.
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Gfi1 functions downstream of Math1 to control intestinal secretory cell subtype allocation and differentiation

TL;DR: It is demonstrated that Gfi1 functions downstream of Math1 in intestinal secretory lineage differentiation, and a model of intestinal cell fate choice in which beta-catenin and Cdx function upstream ofMath1, and lineage-specific genes such as Ngn3 act downstream of Gfi2 is proposed.
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Tryptophan Biosynthesis Protects Mycobacteria from CD4 T-Cell-Mediated Killing

TL;DR: A genome-scale study of Mycobacterium tuberculosis is presented that uncovers the bacterial determinants of surviving host immunity, sets of genes the authors term "counteractomes", and identifies a small-molecule inhibitor of Mtb tryptophan synthesis, which converts Mtb into a tryPTophan auxotroph and restores the efficacy of a failed host defense.
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A common variant of the latrophilin 3 gene, LPHN3, confers susceptibility to ADHD and predicts effectiveness of stimulant medication

TL;DR: Functional studies revealed that LPHN3 variants are expressed in key brain regions related to attention and activity, affect metabolism in neural circuits implicated in ADHD, and are associated with response to stimulant medication.