D
Dennis B. Lubahn
Researcher at University of Missouri
Publications - 201
Citations - 29869
Dennis B. Lubahn is an academic researcher from University of Missouri. The author has contributed to research in topics: Estrogen receptor & Estrogen receptor alpha. The author has an hindex of 79, co-authored 200 publications receiving 28809 citations. Previous affiliations of Dennis B. Lubahn include Mayo Clinic & University of Missouri System.
Papers
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Journal ArticleDOI
Estrogen inhibits the vascular injury response in estrogen receptor alpha-deficient mice.
Mark D. Iafrati,Richard H. Karas,Mark Aronovitz,Sung K. Kim,Theodore R. Sullivan,Dennis B. Lubahn,Thomas F. O'Donnell,Kenneth S. Korach,Michael E. Mendelsohn +8 more
TL;DR: It is demonstrated that estrogen inhibits vascular injury by a novel mechanism that is independent of the classic estrogen receptor, ERα, which inhibits the development of atherosclerotic and injury-induced vascular lesions.
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Immunohistochemical Localization of the Androgen Receptor in Rat and Human Tissues
TL;DR: The results of these studies indicate that AR can be detected immunohistochemically in a variety of tissues and cell types using antipeptide polyclonal antibodies.
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Roles of estrogen receptor-alpha gene expression in reproduction-related behaviors in female mice.
TL;DR: Findings suggest that ER alpha gene expression plays a key role in female mice, not only for sexual behavior but also for other interrelated behaviors, such as parental and aggressive behaviors.
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Identification of androgen receptors in normal human osteoblast-like cells
D. S. Colvard,Erik Fink Eriksen,Philip E. Keeting,E M Wilson,Dennis B. Lubahn,F S French,B L Riggs,Thomas C. Spelsberg +7 more
TL;DR: It is concluded that both androgens and estrogens act directly on human bone cells through their respective receptor-mediated mechanisms.
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Behavioral effects of estrogen receptor gene disruption in male mice
TL;DR: In this paper, the role of estrogen receptor activation by endogenous estrogen in the development of male-typical behaviors was determined by the use of transgenic estrogen-receptor-deficient (ERKO) mice.