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Edwin H. Cook
Researcher at University of Illinois at Chicago
Publications - 339
Citations - 61045
Edwin H. Cook is an academic researcher from University of Illinois at Chicago. The author has contributed to research in topics: Autism & Heritability of autism. The author has an hindex of 102, co-authored 337 publications receiving 54518 citations. Previous affiliations of Edwin H. Cook include University of Chicago & University of Illinois at Urbana–Champaign.
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Significant neuronal soma volume deficit in the limbic system in subjects with 15q11.2-q13 duplications
Jerzy Wegiel,Michael Flory,N. Carolyn Schanen,Edwin H. Cook,Krzysztof Nowicki,Izabela Kuchna,Humi Imaki,Shuang Yong Ma,Jarek Wegiel,Eric London,Manuel F. Casanova,Thomas Wisniewski,W. Ted Brown +12 more
TL;DR: This study demonstrates that alterations in the trajectory of neuronal growth throughout the lifespan are a core pathological features of idiopathic and syndromic autism.
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Are the arginine vasopressin V1a receptor microsatellites related to hypersexuality in children with a prepubertal and early adolescent bipolar disorder phenotype
TL;DR: Examining family-based transmission of the number of 5' flanking arginine vasopressin V1a receptor (AVPR1A) microsatellites to hypersexual and uninhibited people-seeking probands with a prepubertal and early adolescent bipolar disorder phenotype was not supported.
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Commentary on “Platelet Studies in Autism Spectrum Disorder Patients and First-Degree Relatives”
George M. Anderson,Edwin H. Cook +1 more
TL;DR: The results need a fuller discussion in the context of prior studies, think that certain aspects of the interpretation need to be reassessed, and attempt to provide a framework for further research in this area.
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Reduction of increased repetitive self‐grooming in ASD mouse model by metabotropic 5 glutamate receptor antagonism; randomized controlled trial of early start denver model
TL;DR: The somewhat unexpected finding of reduced self-grooming in the study by Silverman and colleagues being reviewed suggests the importance of including outcome measures for restricted and repetitive behaviors as well as social and learning measures in the clinical trials of mGluR5 antagonists.
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The Werner’s syndrome 4330T>C (Cys1367Arg) gene variant does not affect the in vitro cytotoxicity of topoisomerase inhibitors and platinum compounds
Federico Innocenti,Snezana Mirkov,Ramamoorthy Nagasubramanian,Jacqueline Ramírez,Wanqing Liu,Wasim K. Bleibel,Sunita J. Shukla,Kathleen A. Hennessy,Gary L. Rosner,Edwin H. Cook,M. Eileen Dolan,Mark J. Ratain +11 more
TL;DR: Results suggest that this nonsynonymous variant in the WS gene (WRN) has relatively normal function at the cellular level.