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Gary M. Brittenham

Researcher at Columbia University

Publications -  262
Citations -  17412

Gary M. Brittenham is an academic researcher from Columbia University. The author has contributed to research in topics: Anemia & Iron deficiency. The author has an hindex of 62, co-authored 252 publications receiving 16156 citations. Previous affiliations of Gary M. Brittenham include Columbia University Medical Center & MetroHealth.

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Journal ArticleDOI

High-dose carbonyl iron for iron deficiency anemia: a randomized double-blind trial.

Victor R. Gordeuk, +4 more
- 01 Dec 1987 - 
TL;DR: The results suggest that the principal advantage to the use of carbonyl Fe would derive from its safety rather than from the large doses that can be given.
Journal Article

Efficacy of Artequick versus artesunate-mefloquine in the treatment of acute uncomplicated falciparum malaria in Thailand.

Noppadon Tangpukdee, +9 more
- 01 Jan 2008 - 
TL;DR: It is concluded that Artequick was as effective and well tolerated as artesunate-mefloquine and could be used as an alternative treatment for multidrug-resistant Plasmodium falciparum malaria in Southeast Asia.
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Rapid automated liver quantitative susceptibility mapping.

Ramin Jafari, +13 more
- 01 Sep 2019 - 
TL;DR: This work investigates the feasibility of automated quantitative susceptibility mapping (QSM) to measure the liver iron concentration (LIC) and finds it feasible to guide iron‐chelating therapy for patients with transfusional iron overload.
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Transferrin saturation and recovery from coma in cerebral malaria

Victor R. Gordeuk, +7 more
- 01 Jun 1995 - 
TL;DR: It is concluded that increased transferrin saturations may be associated with delayed recovery from coma during standard therapy for cerebral malaria and that serum iron and total iron binding capacity should be measured in future studies.
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Deferoxamine prevents cardiac hypertrophy and failure in the gerbil model of iron-induced cardiomyopathy.

Tianen Yang, +6 more
- 01 Nov 2003 - 
TL;DR: In the gerbil model of iron overload, concurrent administration of DFO with iron prevents both the development of cardiac hypertrophy and the progressive deterioration in cardiac performance that are produced by chronic iron accumulation.