Geicho Nakatsu

TL;DR: Analysis of the microbial communities in human gut mucosae at different stages of colorectal tumorigenesis suggests that a taxonomically defined microbial consortium is implicated in the development of CRC.

TL;DR: Evidence is provided that the fecal microbiota from patients with CRC can promote tumorigenesis in germ-free mice and mice given a carcinogen, and up-regulation of genes involved in cell proliferation, stemness, apoptosis, angiogenesis, invasiveness, and metastasis are revealed in mice fed with stool from Patients with CRC.

TL;DR: Differences in bacterial interactions across stages of gastric carcinogenesis are identified and significant enrichments and network centralities suggest potentially important roles of P. pneumosintes, S. exigua, P. stomatis, D. micra and S. anginosus in GC progression.

TL;DR: This study identified potential diagnostic bacterial markers that are robust across populations, indicating their potential universal use for non-invasive CRC diagnosis and elucidated the ecological networks and functional capacities of CRC-associated microbiota.

TL;DR: This study revealed CRC-associated mycobiome dysbiosis characterised by altered fungal composition and ecology, signifying that the gut myCobiome might play a role in CRC.