G
Georg Fuellen
Researcher at University of Münster
Publications - 12
Citations - 1921
Georg Fuellen is an academic researcher from University of Münster. The author has contributed to research in topics: Protein family & Gene. The author has an hindex of 9, co-authored 12 publications receiving 1872 citations. Previous affiliations of Georg Fuellen include University of Greifswald & Ruhr University Bochum.
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Journal ArticleDOI
The Bioperl Toolkit: Perl Modules for the Life Sciences
Jason E. Stajich,David Block,David Block,Kris Boulez,Steven E. Brenner,Stephen A. Chervitz,Chris Dagdigian,Georg Fuellen,James G. R. Gilbert,Ian F Korf,Hilmar Lapp,Heikki Lehväslaiho,Chad Matsalla,Christopher J. Mungall,Brian I. Osborne,Matthew Pocock,Peter Schattner,Martin Senger,Lincoln Stein,Elia Stupka,Mark Wilkinson,Ewan Birney +21 more
TL;DR: The overall architecture of the Bioperl toolkit is described, the problem domains that it addresses, and specific examples of how the toolkit can be used to solve common life-sciences problems are given.
Journal ArticleDOI
Absence of S100A12 in mouse: implications for RAGE-S100A12 interaction.
TL;DR: Aprotein that binds to a repeated EP motif in the intracellular region of LAG-3, may participate in the down-regulation of the CD3/TCR activation pathway.
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Cloning, genomic organization, and tissue-specific expression of the RASL11B gene.
Katrin Stolle,Michael Schnoor,Georg Fuellen,Georg Fuellen,Michael Spitzer,Michael Spitzer,Paul Cullen,Stefan Lorkowski +7 more
TL;DR: Results indicate that RASL11B may play a role in TGF-beta1-mediated developmental processes and in pathophysiologies such as inflammation, cancer, and arteriosclerosis.
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Cloning, cellular localization, genomic organization, and tissue-specific expression of the TGFβ1-inducible SMAP-5 gene
Katrin Stolle,Michael Schnoor,Georg Fuellen,Michael Spitzer,Thomas Engel,Friedrich Spener,Paul Cullen,Stefan Lorkowski +7 more
TL;DR: The highest level of expression was found in coronary smooth muscle cells, in which expression of the SMAP-5 gene was induced by transforming growth factor beta1, thus indicating that this protein may play an important role in inflammation.
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Computational searches for missing orthologs: the case of S100A12 in mice.
TL;DR: It is indicated that S100A12 has been lost during rodent evolution, probably due to a deletion, because experimental approaches failed to identify it, and an analysis of gene locus evolution found homology to a region close to the human S 100A9 locus.