G
Gi-Byoung Nam
Researcher at University of Ulsan
Publications - 158
Citations - 3441
Gi-Byoung Nam is an academic researcher from University of Ulsan. The author has contributed to research in topics: Atrial fibrillation & Catheter ablation. The author has an hindex of 26, co-authored 136 publications receiving 2805 citations.
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Journal ArticleDOI
J-Wave syndromes expert consensus conference report: Emerging concepts and gaps in knowledge.
Charles Antzelevitch,Gan-Xin Yan,Michael J. Ackerman,Martin Borggrefe,Domenico Corrado,Jihong Guo,Ihor Gussak,Can Hasdemir,Minoru Horie,Heikki V. Huikuri,Changsheng Ma,Hiroshi Morita,Gi-Byoung Nam,Frédéric Sacher,Wataru Shimizu,Sami Viskin,Arthur A.M. Wilde +16 more
TL;DR: The J-wave syndromes (JWSs) consisting of the Brugada syndrome (BrS) and early repolarization syndrome (ERS) have captured the interest of the cardiology community over the past 2 decades following the identification of BrS as a new clinical entity.
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Mutations in the cardiac L-type calcium channel associated with inherited J-wave syndromes and sudden cardiac death
Elena Burashnikov,Ryan Pfeiffer,Hector Barajas-Martinez,Eva Delpón,Dan Hu,Mayurika Desai,Martin Borggrefe,Michel Haïssaguerre,Ronald J. Kanter,Guido D. Pollevick,Alejandra Guerchicoff,Ruben Laiño,Mark Marieb,Koonlawee Nademanee,Gi-Byoung Nam,Roberto Robles,Rainer Schimpf,Dwight D. Stapleton,Sami Viskin,Stephen L. Winters,Christian Wolpert,Samuel Zimmern,Christian Veltmann,Charles Antzelevitch +23 more
TL;DR: The study results indicate that mutations in the LTCCs are detected in a high percentage of probands with J-wave syndromes associated with inherited cardiac arrhythmias, suggesting that genetic screening of Ca(v) genes may be a valuable diagnostic tool in identifying individuals at risk.
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Augmentation of J Waves and Electrical Storms in Patients with Early Repolarization
TL;DR: To the Editor: Early repolarization, consisting of an elevation of the QRS–ST junction (J point), QRS notching or slurring (J wave), and a tall, symmetric T wave, is generally considered to be benign.
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Mutations in SCN10A are responsible for a large fraction of cases of Brugada syndrome
Dan Hu,Hector Barajas-Martinez,Ryan Pfeiffer,Fabio Dezi,Jenna Pfeiffer,Tapan Buch,Matthew J. Betzenhauser,Luiz Belardinelli,Kristopher M. Kahlig,Sridharan Rajamani,Harry DeAntonio,Robert J. Myerburg,Hiroyuki Ito,Pramod Deshmukh,Mark Marieb,Gi-Byoung Nam,Atul Bhatia,Can Hasdemir,Michel Haïssaguerre,Christian Veltmann,Rainer Schimpf,Martin Borggrefe,Sami Viskin,Charles Antzelevitch +23 more
TL;DR: This study identified SCN10A as a major susceptibility gene for BrS, thus greatly enhancing the ability to genotype and risk stratify probands and family members.
Journal ArticleDOI
Mode of onset of ventricular fibrillation in patients with early repolarization pattern vs. Brugada syndrome.
Gi-Byoung Nam,Kwan-Ho Ko,Jun Kim,Kyoung-Min Park,Kyoung-Suk Rhee,Kee-Joon Choi,You-Ho Kim,Charles Antzelevitch +7 more
TL;DR: This study provides additional evidence in support of the hypothesis that ER pattern in the ECG is not always benign and transient augmentation of global J waves may be indicative of a highly arrhythmogenic substrate heralding multiple episodes of VF in patients with ER pattern.