scispace - formally typeset
G

Glyn Lewis

Researcher at University College London

Publications -  768
Citations -  57050

Glyn Lewis is an academic researcher from University College London. The author has contributed to research in topics: Population & Longitudinal study. The author has an hindex of 113, co-authored 734 publications receiving 49316 citations. Previous affiliations of Glyn Lewis include University College Hospital & St Bartholomew's Hospital.

Papers
More filters
Journal Article

Deciding who gets treatment for depression and anxiety: a study of consecutive GP attenders

TL;DR: In this article, the authors investigated factors associated with receiving active treatment among consecutive attenders identified by GPs as having a common mental disorder, including higher symptom score, as measured by the General Health Questionnaire (GHQ) (odds ratio [OR] = 1.09; 95% confidence interval [CI] =1.06 to 1.13; P P P = 0.006), were both associated with an increased likelihood of being offered active treatment.
Journal ArticleDOI

Pain in veterans of the Gulf War of 1991: a systematic review

TL;DR: A higher proportion of veterans of the Persian Gulf War of 1991 reported symptoms of pain than military comparison groups, consistent with previously demonstrated increased reporting of more general symptoms in these veterans compared with non-Gulf military groups.
Journal ArticleDOI

Dimensions of Parental Alcohol Use/Problems and Offspring Temperament, Externalizing Behaviors, and Alcohol Use/Problems

TL;DR: Self-reported levels of maternal AC during pregnancy uniquely predicted externalizing behaviors in childhood and adolescence, and maternal and paternal AC and AP at age 18 were more strongly predicted by parental factors than at age 15.
Journal ArticleDOI

Exploring neural dysfunction in 'clinical high risk' for psychosis: a quantitative review of fMRI studies.

TL;DR: A comprehensive review of fMRI studies was carried out to observe for general trends with respect to brain regions and cognitive systems most likely to be dysfunctional in CHR individuals, and implicated four key dysfunctional and distinct neural regions in the CHR group.