H
Haruko Masumiya
Researcher at Hyogo College of Medicine
Publications - 36
Citations - 1290
Haruko Masumiya is an academic researcher from Hyogo College of Medicine. The author has contributed to research in topics: Chronotropic & Efonidipine. The author has an hindex of 17, co-authored 36 publications receiving 1180 citations. Previous affiliations of Haruko Masumiya include Tokyo Medical and Dental University & Toho University.
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Journal ArticleDOI
MG53 nucleates assembly of cell membrane repair machinery
Chuanxi Cai,Haruko Masumiya,Noah Weisleder,Noriyuki Matsuda,Miyuki Nishi,Moonsun Hwang,Jae-Kyun Ko,Pei-Hui Lin,Angela Thornton,Xiaoli Zhao,Zui Pan,Shinji Komazaki,Marco Brotto,Hiroshi Takeshima,Jianjie Ma +14 more
TL;DR: It is reported that MG53, a muscle-specific tripartite motif family protein (TRIM72), is a component of the sarcolemmal membrane-repair machinery and that MG 53 may initiate the assembly of the membrane repair machinery in an oxidation-dependent manner.
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MG53 Regulates Membrane Budding and Exocytosis in Muscle Cells
Chuanxi Cai,Haruko Masumiya,Noah Weisleder,Zui Pan,Miyuki Nishi,Miyuki Nishi,Shinji Komazaki,Hiroshi Takeshima,Hiroshi Takeshima,Jianjie Ma +9 more
TL;DR: The identified tripartite motif (TRIM72) family member protein named MG53 is defined and its role in mediating the dynamic process of membrane fusion and exocytosis in striated muscle is defined.
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Inhibition of myocardial L- and T-type Ca2+ currents by efonidipine: possible mechanism for its chronotropic effect.
TL;DR: Efonidipine was shown to have inhibitory effects on both L- and T-type Ca2+ currents, which may contribute to its high negative chronotropic potency.
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Developmental changes in action potential and membrane currents in fetal, neonatal and adult guinea-pig ventricular myocytes
TL;DR: The electrophysiological properties of the guinea-pig ventricular myocytes were demonstrated to change during pre- and postnatal development, and the observed changes in action potential duration could be explained by changes in the balance between lK and lCaL.
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Functional uncoupling between Ca2+ release and afterhyperpolarization in mutant hippocampal neurons lacking junctophilins
Shigeki Moriguchi,Miyuki Nishi,Shinji Komazaki,Hiroyuki Sakagami,Taisuke Miyazaki,Haruko Masumiya,Shin ya Saito,Masahiko Watanabe,Hisatake Kondo,Hiromu Yawo,Kohji Fukunaga,Hiroshi Takeshima +11 more
TL;DR: It is proposed that in JP-DKO neurons lacking afterhyperpolarization, the functional communications between NMDA receptors, ryanodine receptors, and small-conductance Ca(2+)-activated K(+) channels are disconnected because of JMC disassembly, and JPs seem to have an essential role in neural excitability fundamental to plasticity and integrated functions.