Harvey J. Grill

TL;DR: The results show that leptin can change body fat independent of a change in food intake or energy expenditure, that the forebrain normally prevents leptin from inhibiting energy expenditure through mechanisms initiated in the caudal brainstem or peripheral tissues, and that the leptin response in both intact and CD rats is determined by the energy status of the animal.

TL;DR: Data demonstrate that, in neural isolation from the hypothalamus, cold exposure drives caudal brain stem neuronal activity and engages local effectors that trigger sympathetic energetic and cardiac responses that are comparable in many, but not in all, respects to those seen in neurologically intact rats.

TL;DR: The view that excitatory hindbrain–forebrain projections are necessary for cisplatin's untoward effects on energy intake is supported, elucidating a key neuroanatomical circuit driving pathological anorexia and weight loss that accompanies chemotherapy treatment.

TL;DR: These studies are the first to show that hindbrain administration of leptin or a melanocortin receptor agonist alters energy balance in mice likely via participation of hindbrain POMC neurons.

TL;DR: In this paper, a temporal pattern analysis based on the spout-licking literature is applied to the rhythmic movements that attend intraoral infusion of fluids normally ingested by rats, which can be likened to spout licking in several respects, including the frequency of bursts, mean burst duration, pause durations, coefficient of variation for the distribution of within-burst intermovement intervals.