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Harvey J. Grill

Researcher at University of Pennsylvania

Publications -  172
Citations -  15156

Harvey J. Grill is an academic researcher from University of Pennsylvania. The author has contributed to research in topics: Leptin & Forebrain. The author has an hindex of 63, co-authored 168 publications receiving 13834 citations. Previous affiliations of Harvey J. Grill include Beth Israel Deaconess Medical Center & Rockefeller University.

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Fourth ventricular phlorizin dissociates feeding from hyperglycemia in rats

TL;DR: The glucodynamic action of phlorizin appears selective for that mechanism mediating the behavioral compensatory response of feeding in contrast to the dual effects produced by 5-TG, which suggests that different caudal brainstem mechanisms control behavioral and autonomic compensatory responses.
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Behavioral processes underlying the intake suppressive effects of melanocortin 3/4 receptor activation in the rat

TL;DR: Results suggest thatMTII reduces intake by amplifying post-ingestive feedback inhibition, and that MTII-treated rats increase the number of licks emitted in response to the lick volume reduction discounts the suggestion that intake inhibition is secondary to disruption of motor performance.
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Protein-tyrosine Phosphatase 1B (PTP1B) Is a Novel Regulator of Central Brain-derived Neurotrophic Factor and Tropomyosin Receptor Kinase B (TrkB) Signaling

TL;DR: It is shown that the tropomyosin receptor kinase B (TrkB) receptor is a direct PTP1B substrate and that enhanced BDNF/TrkB signaling may contribute to the beneficial metabolic effects of PTP 1B deficiency.
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Conditioned reversal of reactions to normally avoided tastes.

TL;DR: It is demonstrated here that when a normally avoided taste (quinine or HCl) signals a preferred taste (sucrose), the oral motor responses gradually undergo two types of change as a function of trials, including aversive and ingestive responses.
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Overfeeding-induced weight gain suppresses plasma ghrelin levels in rats

TL;DR: Having discounted diet composition and differences in gastric contents at the time of blood sampling, the decrease in ghrelin levels with overfeeding can be unambiguously attributed to physiological correlates of weight gain.