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Harvey J. Grill

Researcher at University of Pennsylvania

Publications -  172
Citations -  15156

Harvey J. Grill is an academic researcher from University of Pennsylvania. The author has contributed to research in topics: Leptin & Forebrain. The author has an hindex of 63, co-authored 168 publications receiving 13834 citations. Previous affiliations of Harvey J. Grill include Beth Israel Deaconess Medical Center & Rockefeller University.

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Regional influence of cocaine on evoked dopamine release in the nucleus accumbens core: A role for the caudal brainstem.

TL;DR: The data demonstrate that cocaine-induced effects on phasic dopamine signaling are mediated via actions throughout the brain including the hindbrain, and are the first to show that hindbrain (fourth ventricle)-delivered cocaine modulates phasIC dopamine signaling.
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New horizons for future research – Critical issues to consider for maximizing research excellence and impact

TL;DR: Wolfgang Langhans *, Roger Adan , Myrtha Arnold , William A. Banks , J. Patrick Card, Megan J. Moran, Gustavo Pacheco-López, Deepti Ramachandran, Helen Raybould, Linda Rinaman, Willis K. Samson, Graciela Sanchez-Watts, Randy J. Watts.
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Central leptin signaling transmits positive valence

TL;DR: It is shown that centrally-delivered leptin reduced food intake and conditioned a place preference in food-restricted as well as ad libitum fed rats, suggesting that leptin signaling is intrinsically preferred, and support the view that signals of energy surfeit are associated with positive affect.
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The Mesencephalic Trigeminal Nucleus Controls Food Intake and Body Weight via Hindbrain POMC Projections.

TL;DR: In this paper, the mesencephalic trigeminal nucleus (Mes5) was found to express the melanocortin-4 receptor and microinjection of the MC4R agonist melanotan-II (MTII) suppresses food intake and body weight in the mouse.
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Individual Differences in Behavioral Responses to Palatable Food or to Cholecystokinin Predict Subsequent Diet-Induced Obesity.

TL;DR: This study investigated whetherindividual differences in behavioral responses to palatable food and to the satiation signal cholecystokinin in outbred chow‐maintained Sprague‐Dawley rats enabled prediction of individual differences in weight gained after subsequent high‐fat/high‐sugar diet (HFHSD) maintenance.