H
Hermona Soreq
Researcher at Hebrew University of Jerusalem
Publications - 521
Citations - 27215
Hermona Soreq is an academic researcher from Hebrew University of Jerusalem. The author has contributed to research in topics: Acetylcholinesterase & Cholinergic. The author has an hindex of 80, co-authored 503 publications receiving 25411 citations. Previous affiliations of Hermona Soreq include Tel Aviv Sourasky Medical Center & Life Sciences Institute.
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Differentiation of human embryonic stem cells into embryoid bodies compromising the three embryonic germ layers.
Joseph Itskovitz-Eldor,Maya Schuldiner,Dorit Karsenti,Amir Eden,Ofra Yanuka,Michal Amit,Hermona Soreq,Nissim Benvenisty +7 more
TL;DR: The ability to induce formation of human embryoid bodies that contain cells of neuronal, hematopoietic and cardiac origins will be useful in studying early human embryonic development as well as in transplantation medicine.
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Amplification, enhanced expression and possible rearrangement of EGF receptor gene in primary human brain tumours of glial origin
Towia A. Libermann,Harris R. Nusbaum,Nissim Razon,Richard Kris,Irit Lax,Hermona Soreq,Nigel Whittle,Michael D. Waterfield,Axel Ullrich,Joseph Schlessinger +9 more
TL;DR: 4 of 10 primary brain tumours of glial origin which express levels of EGF receptors that are higher than normal also have amplified EGF receptor genes, suggesting that such altered expression and amplification is a particular feature of certain human tumours.
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Function of alternative splicing.
Stefan Stamm,Shani Ben-Ari,Ilona Rafalska,Yesheng Tang,Zhaiyi Zhang,Debra Toiber,Thangavel Alphonse Thanaraj,Hermona Soreq +7 more
TL;DR: Evidence is now accumulating that alternative splicing coordinates physiologically meaningful changes in protein isoform expression and is a key mechanism to generate the complex proteome of multicellular organisms.
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Acetylcholinesterase New roles for an old actor
Hermona Soreq,Shlomo Seidman +1 more
TL;DR: The time is ripe to summarize the evidence on a remarkable diversity of acetylcholinesterase functions, as well as some of the long-suspected 'non-classical' actions of this enzyme, which have more recently driven a profound revolution in research.
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Acute stress facilitates long-lasting changes in cholinergic gene expression
TL;DR: The results suggest a model in which robust cholinergic stimulation triggers rapid induction of the gene encoding the transcription factor c-Fos, which mediates selective regulatory effects on the long-lasting activities of genes involved in acetylcholine metabolism.