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Isabel M. Smith

Researcher at Dalhousie University

Publications -  161
Citations -  10790

Isabel M. Smith is an academic researcher from Dalhousie University. The author has contributed to research in topics: Autism & Autism spectrum disorder. The author has an hindex of 42, co-authored 137 publications receiving 9179 citations. Previous affiliations of Isabel M. Smith include Izaak Walton Killam Health Centre & University of Minnesota.

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Executive function in preschoolers: A review using an integrative framework.

TL;DR: The authors focus on 3 EF components: working memory, response inhibition, and shifting and conceive of the central executive as a central attention system that is involved in all EF component operations.
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Whole genome sequencing resource identifies 18 new candidate genes for autism spectrum disorder

Ryan K. C. Yuen, +89 more
- 06 Mar 2017 - 
TL;DR: Se sequencing of 5,205 samples from families with ASD, accompanied by clinical information, creating a database accessible on a cloud platform and through a controlled-access internet portal that identified 18 new candidate ASD-risk genes and found that participants bearing mutations in susceptibility genes had significantly lower adaptive ability.
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Risperidone in the Treatment of Disruptive Behavioral Symptoms in Children With Autistic and Other Pervasive Developmental Disorders

TL;DR: Risperidone was well tolerated and efficacious in treating behavioral symptoms associated with PDD in children and seemed manageable with dose/dose-schedule modification.
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Early language and communication development of infants later diagnosed with autism spectrum disorder.

TL;DR: It is confirmed that delays in communication and language development are apparent early in life in children with ASD, and developmental surveillance should include monitoring for delays in gesture, which may be among the earliest signs of ASD.
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Imitation and action in autism: A critical review.

TL;DR: The evidence for an imitative deficit in autism and for the possible role of deficiencies in the representation of actions are considered and an argument is developed for the claim that the imitation problem is diagnostic of a basic information-processing rather than a social dysfunction.