J
J. van der Velden
Researcher at VU University Amsterdam
Publications - 90
Citations - 3936
J. van der Velden is an academic researcher from VU University Amsterdam. The author has contributed to research in topics: Vulvar cancer & Hypertrophic cardiomyopathy. The author has an hindex of 30, co-authored 90 publications receiving 3274 citations. Previous affiliations of J. van der Velden include Netherlands Cancer Institute & VU University Medical Center.
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Journal ArticleDOI
ESMO-ESGO consensus conference recommendations on ovarian cancer: pathology and molecular biology, early and advanced stages, borderline tumours and recurrent disease
Nicoletta Colombo,Cristiana Sessa,A. du Bois,Jonathan A. Ledermann,WG McCluggage,Iain A. McNeish,P. Morice,Sandro Pignata,Isabelle Ray-Coquard,Ignace Vergote,Thaïs Baert,I. Belaroussi,A. Dashora,Siel Olbrecht,François Planchamp,Denis Querleu,Susana Banerjee,P. Blecharz,Ilan Bruchim,David Cibula,Nicole Concin,Ben Davidson,Mojgan Devouassoux-Shisheboran,Annamaria Ferrero,R.M. Glasspool,Antonio González-Martín,Viola Heinzelmann-Schwarz,Florence Joly,J. W. Kim,Frédéric Kridelka,Domenica Lorusso,Sven Mahner,Mikio Mikami,Mansoor Raza Mirza,Shibani Nicum,Dearbhaile M. O'Donnell,Patricia Pautier,Alexandros Rodolakis,Jalid Sehouli,Fatih Selcukbiricik,Naveena Singh,David S.P. Tan,Dirk Timmerman,Germana Tognon,J. van der Velden,Petronella O. Witteveen,AG Zeimet +46 more
TL;DR: The recommendations presented here are based on the best available evidence and expert agreement and are presented with a summary of evidence supporting each recommendation.
Journal ArticleDOI
Comorbidity of chronic diseases in general practice
TL;DR: The aim of this study is to describe the extent of comorbidity for the following diseases: hypertension, chronic ischemic heart disease, diabetes mellitus, chronic nonspecific lung disease, osteoarthritis.
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Increased Ca2+-sensitivity of the contractile apparatus in end-stage human heart failure results from altered phosphorylation of contractile proteins
J. van der Velden,Zoltán Papp,R. Zaremba,Nicky M. Boontje,J. W. de Jong,V. J. Owen,P. B.J. Burton,P. Goldmann,Kornelia Jaquet,Ger J.M. Stienen +9 more
TL;DR: The increased Ca(2+)-responsiveness of the contractile apparatus in end-stage failing human hearts cannot be explained by a shift in contractile protein isoforms, but results from the complex interplay between changes in the phosphorylation status of MLC-2 and TnI.
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Sentinel nodes in vulvar cancer: Long-term follow-up of the GROningen INternational Study on Sentinel nodes in Vulvar cancer (GROINSS-V) I.
te Nienke Grootenhuis,van der Ate Zee,H. C. van Doorn,J. van der Velden,Ignace Vergote,Vanna Zanagnolo,Peter Baldwin,Katja N. Gaarenstroom,E. Van Dorst,J. W. Trum,Brigitte F. M. Slangen,Ingo B. Runnebaum,Karl Tamussino,Ralph H. Hermans,Diane Provencher,G. H. de Bock,J.A. de Hullu,Maaike H. M. Oonk +17 more
TL;DR: Long-term follow-up of patients with unifocal squamous cell carcinoma of the vulva included in GROINSS-V-I showed that application of the SN-procedure in patients with early-stage vulvar cancer is safe, and 10-year disease-specific survival decreased from 90% for Patients without to 69% for patients with a local recurrence.
Journal ArticleDOI
The effect of myosin light chain 2 dephosphorylation on Ca2+-sensitivity of force is enhanced in failing human hearts
J. van der Velden,Zoltán Papp,Nicky M. Boontje,R. Zaremba,J. W. de Jong,Paul M.L. Janssen,Gerd Hasenfuss,Ger J.M. Stienen +7 more
TL;DR: Surprisingly, the contractile response to MLC-2 dephosphorylation is enhanced in failing hearts, despite the reduced level of basal M LC-2 phosphorylation, suggesting a potential compensatory mechanism to reverse the detrimental effects of increased Ca( 2+)-sensitivity and impaired Ca(2+)-handling on diastolic function in human heart failure.