J
Jacob Raber
Researcher at Oregon Health & Science University
Publications - 253
Citations - 13602
Jacob Raber is an academic researcher from Oregon Health & Science University. The author has contributed to research in topics: Apolipoprotein E & Water maze. The author has an hindex of 53, co-authored 230 publications receiving 11862 citations. Previous affiliations of Jacob Raber include Scripps Health & University of California, San Francisco.
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Enhanced ethanol-, but not cocaine-induced, conditioned place preference in Apoe(-/-) mice.
TL;DR: It is suggested that apoE normally reduces the conditioned rewarding properties of ethanol but not of cocaine, and data support a possible role for apeE in modulating the conditioned rewards of ethanol.
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ApoE isoform-dependent deficits in extinction of contextual fear conditioning.
TL;DR: Clinical data showing an association of apoE2 with susceptibility to specific symptom clusters in PTSD supports an important role for apoS isoform in the extinction of conditioned fear, and acquisition, consolidation, and extinction in human and animal models.
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16)Oxygen irradiation enhances cued fear memory in B6D2F1 mice.
TL;DR: Cued fear memory was significantly stronger in mice irradiated with (16)O ions at a dose of 0.4 or 0.8 Gy than in sham-irradiated mice or following irradiation at 1.6 Gy.
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Fetal domoic acid exposure affects lateral amygdala neurons, diminishes social investigation and alters sensory-motor gating.
TL;DR: The hypothesis that prenatal exposure of FVB mice to low levels of DA would result in diminished social interaction and sensory motor gating associated with alterations in parvalbumin immunoreactivity in relevant brain regions undergoing development during and following DA exposure was tested.
Journal ArticleDOI
Effects of 56Fe radiation on hippocampal function in mice deficient in chemokine receptor 2 (CCR2)
Jacob Raber,Antiño R. Allen,Susanna Rosi,Sourabh Sharma,Catherine A. Dayger,Matthew J. Davis,John R. Fike +6 more
TL;DR: While measures of neurogenesis and gliogenesis appeared to be modulated by CCR2, there were no effects of genotype on the total numbers of newly born activated microglia before or after irradiation, indicating that other mechanisms are involved in the genotype-dependent radiation response.