Jeffery K. Taubenberger

TL;DR: It is proposed that AIV in wild birds forms transient “genome constellations,” continually reshuffled by reassortment, in contrast to the spread of a limited number of stable genome constellation that characterizes the evolution of mammalian-adapted influenza A viruses.

TL;DR: Postmortem lung sections from 8 patients who died from SARS during the spring 2003 Singapore outbreak studied, finding the predominant pattern of lung injury in all 8 cases was diffuse alveolar damage, and the histology varied according to the duration of illness.

TL;DR: The pathology of human influenza has been studied most intensively during the three pandemics of the last century, but it is important to revisit this subject because of the recent emergence of avian H5N1 influenza in humans as well as the threat of a new pandemic.

TL;DR: It is demonstrated that an A/H1N1 isolate from the 1947 epidemic acquired novel PB2 and HA genes through intra-subtype reassortment, which may explain the abrupt antigenic evolution of this virus.

TL;DR: Influenza A viruses infect large numbers of warm-blooded animals, including wild birds, domestic birds, pigs, horses, and humans, and can switch hosts to form new lineages in novel hosts.