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Jeffrey F. Harper

Researcher at University of Nevada, Reno

Publications -  112
Citations -  17859

Jeffrey F. Harper is an academic researcher from University of Nevada, Reno. The author has contributed to research in topics: Arabidopsis & Calmodulin. The author has an hindex of 62, co-authored 106 publications receiving 16407 citations. Previous affiliations of Jeffrey F. Harper include Scripps Research Institute.

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Transcriptome Changes for Arabidopsis in Response to Salt, Osmotic, and Cold Stress

TL;DR: Together results from all three stresses identified 2,409 genes with a greater than 2-fold change over control, suggesting that about 30% of the transcriptome is sensitive to regulation by common stress conditions, and supporting the hypothesis that an important function of the circadian clock is to “anticipate” predictable stresses such as cold nights.
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Phylogenetic Relationships within Cation Transporter Families of Arabidopsis

TL;DR: This analysis has focused on cation transporter gene families for which initial characterizations have been achieved for individual members, including potassium transporters and channels, sodium transporter, calcium antiporters, cyclic nucleotide-gated channels, cation diffusion facilitator proteins, natural resistance-associated macrophage proteins, and Zn-regulated transporter Fe-regulatedporter-like proteins.
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Calcium at the Crossroads of Signaling

TL;DR: Calcium Signals are identified as a central Paradigm in Stimulus–Response Coupling and are normally composed of elements that include calcium and Na6(SO4)2, Na2SO4, and Na2CO3.
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Communicating with calcium

TL;DR: Calcium as a Ubiquitous Signal in Plants uses a network of signal transduction pathways to conduct developmental programs, obtain nutrients, control their metabolism, and cope with their environment.
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The Arabidopsis CDPK-SnRK Superfamily of Protein Kinases

TL;DR: Analysis of intron placements supports the hypothesis that CDPKs, CRks, PPCKs and PEPRKs have a common evolutionary origin; however there are no conserved intron positions between these kinases and the SnRK subgroup.