K
Katarina Trajkovic
Researcher at University of Split
Publications - 20
Citations - 3643
Katarina Trajkovic is an academic researcher from University of Split. The author has contributed to research in topics: Endosome & Exocytosis. The author has an hindex of 8, co-authored 19 publications receiving 2999 citations. Previous affiliations of Katarina Trajkovic include University of Tübingen & Max Planck Society.
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Journal ArticleDOI
Ceramide Triggers Budding of Exosome Vesicles into Multivesicular Endosomes
Katarina Trajkovic,Chieh Hsu,Salvatore Chiantia,Lawrence Rajendran,Dirk Wenzel,Felix T. Wieland,Petra Schwille,Britta Brügger,Mikael Simons +8 more
TL;DR: It is found that cargo is segregated into distinct subdomains on the endosomal membrane and that the transfer of exosome-associated domains into the lumen of theendosome did not depend on the function of the ESCRT (endosomal sorting complex required for transport) machinery, but required the sphingolipid ceramide.
Journal ArticleDOI
Neuron-glia communication in the control of oligodendrocyte function and myelin biogenesis.
Mikael Simons,Katarina Trajkovic +1 more
TL;DR: Interconnections at multiple levels show how neurons and glia cooperate to build a complex network during development.
Journal ArticleDOI
Neuron to glia signaling triggers myelin membrane exocytosis from endosomal storage sites.
Katarina Trajkovic,Ajit Singh Dhaunchak,José T. Goncalves,Dirk Wenzel,Anja Schneider,Anja Schneider,Gertrude Bunt,Klaus-Armin Nave,Mikael Simons +8 more
TL;DR: It is shown that neurons regulate myelin membrane trafficking in oligodendrocytes, and a cAMP-dependent neuronal signal triggers the transport of PLP from LEs/Ls to the plasma membrane, revealing a fundamental and novel role of LEs/, to store and release PLP in a regulated fashion.
Journal ArticleDOI
Parkin Modulates Endosomal Organization and Function of the Endo-Lysosomal Pathway
TL;DR: A role is established for Parkin in regulating the endo-lysosomal pathway and retromer function and raise the possibility that alterations in these pathways contribute to the development of pathology in Parkin-linked Parkinson's disease.
Journal ArticleDOI
Hyperphosphorylation and Aggregation of Tau in Experimental Autoimmune Encephalomyelitis
Anja Schneider,Anja Schneider,Gilda Wright Araújo,Katarina Trajkovic,Katarina Trajkovic,Martin M. Herrmann,Doron Merkler,Eva Maria Mandelkow,Robert Weissert,Mikael Simons,Mikael Simons +10 more
TL;DR: Analysis of tau phosphorylation, solubility and distribution in the brainstem of rats with experimental autoimmune encephalomyelitis provides evidence that axonal damage in EAE and possibly MS is linked to tau pathology.