Katherine C. Michelis

TL;DR: It is shown that EndMT contributes to atherosclerotic patho-biology and is associated with complex plaques that may be related to clinical events, and the extent of EndMT correlates with an unstable plaque phenotype, which appears driven by altered collagen-MMP production in EndMT-derived cells.

TL;DR: In this Article, the catalogue number for the anti-Fap-Alexa Fluor 647 antibody is listed incorrectly and should have read bs-5758R-A647 instead of b s-5760R- a647.

TL;DR: The clinical course of FMD involving the coronary arteries is reviewed, guidance for diagnosis and treatment strategies are provided and a conservative approach is generally preferred.

TL;DR: FMD patients exhibit a plasma proteogenomic and lipid signature that includes potential causative disease drivers, and which holds promise for developing a blood-based test for this disease.

TL;DR: P Phenotypic and transcriptomic profiling, as well as adoptive transfer experiments, revealed a potential role in vascular disease pathogenesis, with the transcriptomic disease signature of these cells being represented in an aortic regulatory gene network that is operative in atherosclerosis.