scispace - formally typeset
K

Katy Janvier

Researcher at French Institute of Health and Medical Research

Publications -  22
Citations -  1231

Katy Janvier is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Tetherin & Endosome. The author has an hindex of 16, co-authored 21 publications receiving 1161 citations. Previous affiliations of Katy Janvier include Centre national de la recherche scientifique & University of Paris.

Papers
More filters
Journal ArticleDOI

Polarized trafficking and surface expression of the AQP4 water channel are coordinated by serial and regulated interactions with different clathrin-adaptor complexes.

TL;DR: It is shown that two independent C‐terminal signals determine AQP4 basolateral membrane targeting in epithelial MDCK cells, and stress‐induced kinase casein kinase (CK)II phosphorylates the Ser276 immediately preceding the tyrosine motif, increasing AQp4–μ3A interaction and enhancing AQP 4–lysosomal targeting and degradation.
Journal ArticleDOI

Targeting of the Human Immunodeficiency Virus Type 1 Envelope to the trans-Golgi Network through Binding to TIP47 Is Required for Env Incorporation into Virions and Infectivity

TL;DR: It is found that the cytoplasmic domain of TMgp41 binds to TIP47, a protein required for the transport of mannose-6-phosphate receptors from endosomes to the TGN, supporting a model in which retrograde transport of Env is implicated in the optimization of fully infectious HIV-1 production.
Journal ArticleDOI

Homo- and heterodimerization of peroxisomal ATP-binding cassette half-transporters.

TL;DR: Results provide the first evidence of both homo- and heterodimerization of mammalian ABC half-transporters and suggest that the loss of ALDP dimerization plays a role in X-ALD pathogenesis.
Journal ArticleDOI

The ESCRT-0 Component HRS is Required for HIV-1 Vpu-Mediated BST-2/Tetherin Down-Regulation

TL;DR: It is demonstrated that Vpu-mediated BST-2/tetherin down-modulation and degradation require HRS (ESCRT-0) function and that knock down of HRS increases cellular levels of BST-1 and restricts virus release, and highlights an additional role for the ESCRT machinery in virus release.
Journal ArticleDOI

Nef-induced Alteration of the Early/Recycling Endosomal Compartment Correlates with Enhancement of HIV-1 Infectivity

TL;DR: It is shown that Nef specifically disturbs the morphology of the early/recycling compartment, inducing a redistribution of early endosomal markers and a shortening of the tubular recycling endosome structures.