Klaus Palme

TL;DR: This work shows that five PIN genes collectively control auxin distribution to regulate cell division and cell expansion in the primary root and reveals an interaction network of auxin transport facilitators and root fate determinants that control patterning and growth of the root primordium.

TL;DR: The PIN-FORMED (PIN1) gene was found to encode a 67-kilodalton protein with similarity to bacterial and eukaryotic carrier proteins, and the AtPIN1 protein was detected at the basal end of auxin transport-competent cells in vascular tissue.

TL;DR: It is shown that auxin accumulates asymmetrically during differential growth in an efflux-dependent manner and that actin-dependent relocalization of PIN3 in response to gravity provides a mechanism for redirecting auxin flux to trigger asymmetric growth.

TL;DR: An ImageJ plugin is presented that enables non-machine-learning experts to analyze their data with U-Net on either a local computer or a remote server/cloud service.

TL;DR: It is shown that the seemingly static localization of PIN1 results from rapid actin-dependent cycling between the plasma membrane and endosomal compartments, suggesting that PIN1 cycling is of central importance for auxin transport and that Auxin transport inhibitors affect efflux by generally interfering with membrane-trafficking processes.