K
Korbinian Kienle
Researcher at Max Planck Society
Publications - 3
Citations - 278
Korbinian Kienle is an academic researcher from Max Planck Society. The author has contributed to research in topics: Swarming (honey bee) & Inflammation. The author has an hindex of 3, co-authored 3 publications receiving 152 citations. Previous affiliations of Korbinian Kienle include University of Freiburg.
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Journal ArticleDOI
Neutrophil swarming: an essential process of the neutrophil tissue response.
TL;DR: It is now clear that intercellular communication among neutrophils amplifies their recruitment in a feed‐forward manner, which provides them with a level of self‐organization during neutrophil swarming, a critical role in maintaining the balance between host protection and inflammation‐driven tissue destruction.
Journal ArticleDOI
Platelet Serotonin Aggravates Myocardial Ischemia/Reperfusion Injury via Neutrophil Degranulation
Maximilian Mauler,Nadine Herr,Claudia Schoenichen,Thilo Witsch,Timoteo Marchini,Carmen Härdtner,Christoph Koentges,Korbinian Kienle,Korbinian Kienle,Véronique Ollivier,Véronique Ollivier,Maximilian Schell,Ludwig Dorner,Christopher Wippel,Daniela Stallmann,Claus Normann,Claus Normann,Heiko Bugger,Paul Walther,Dennis Wolf,Dennis Wolf,Ingo Ahrens,Tim Lämmermann,Benoît Ho-Tin-Noé,Benoît Ho-Tin-Noé,Klaus Ley,Christoph Bode,Ingo Hilgendorf,Daniel Duerschmied +28 more
TL;DR: Serotonin is identified as a potent therapeutic target in neutrophil-dependent thromboinflammation during myocardial reperfusion injury and patients with depression using serotonin reuptake inhibition are presented with suppressed levels of CD11b surface expression on neutrophils and lower myeloperoxidase levels in blood.
Journal ArticleDOI
Neutrophils self-limit swarming to contain bacterial growth in vivo
Korbinian Kienle,Korbinian Kienle,Katharina M. Glaser,Katharina M. Glaser,Sarah Eickhoff,Michael Mihlan,Konrad Knöpper,Eduardo Reátegui,Eduardo Reátegui,Maximilian W. Epple,Maximilian W. Epple,Matthias Gunzer,Matthias Gunzer,Ralf Baumeister,Teresa K. Tarrant,Ronald N. Germain,Daniel Irimia,Wolfgang Kastenmüller,Tim Lämmermann +18 more
TL;DR: This article found that neutrophils have evolved an intrinsic mechanism to self-limit swarming and avoid uncontrolled aggregation during inflammation using GPCR desensitization as a negative feedback control.