M
M. Didier-Bazes
Researcher at French Institute of Health and Medical Research
Publications - 34
Citations - 1292
M. Didier-Bazes is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Subcommissural organ & Glial fibrillary acidic protein. The author has an hindex of 19, co-authored 34 publications receiving 1248 citations.
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Journal ArticleDOI
Spatiotemporal expression patterns of metalloproteinases and their inhibitors in the postnatal developing rat cerebellum.
TL;DR: Regional and cell-specific expression of MMPs and TIMPs closely reflects the successive stages of cerebellar development, thereby suggesting a pivotal role for ECM proteolysis in brain development and plasticity.
Journal ArticleDOI
Papillary Tumor of the Pineal Region
Anne Jouvet,François Fauchon,Pawel P. Liberski,Ghislaine Saint-Pierre,M. Didier-Bazes,Anna Heitzmann,Marie-Bernadette Delisle,Homa Adle Biassette,Sylvie Vincent,Jacqueline Mikol,Nathalie Streichenberger,Samir Ahboucha,Christine Brisson,Marie-Françoise Belin,Michelle Fèvre-Montange +14 more
TL;DR: The morphofunctional features of these papillary tumors of the pineal region, remarkably uniform within this series, are similar to those described for ependymal cells of the subcommissural organ, and the papillary tumor may be derived from these specialized ependylocytes.
Journal ArticleDOI
How to drain without lymphatics? Dendritic cells migrate from the cerebrospinal fluid to the B-cell follicles of cervical lymph nodes
Eric Hatterer,Nathalie Davoust,M. Didier-Bazes,Carine Vuaillat,Christophe Malcus,Marie-Françoise Belin,Serge Nataf +6 more
TL;DR: It is found that dendritic cells injected within brain parenchyma migrate little from their site of injection and do not reach cervical lymph nodes, while intra-CSF-injected DCs either reach cervicalymph nodes or infiltrate the subventricular zone, where neural stem cells reside.
Journal ArticleDOI
Glutamate metabolism is down-regulated in astrocytes during experimental allergic encephalomyelitis.
Hélène Hardin-Pouzet,Michelle Krakowski,Lyne Bourbonnière,M. Didier-Bazes,Elise Tran,Trevor Owens +5 more
TL;DR: It is proposed that the decreased capacity of astrocytes to metabolize glutamate may contribute to EAE pathology because elevated levels of glutamate may be neurotoxic.
Journal ArticleDOI
Human T-cell lymphotropic virus type 1-infected T lymphocytes impair catabolism and uptake of glutamate by astrocytes via Tax-1 and tumor necrosis factor alpha.
Raphaël Szymocha,Hideo Akaoka,Magali Dutuit,Christophe Malcus,M. Didier-Bazes,Marie-Françoise Belin,Pascale Giraudon +6 more
TL;DR: Tax-1 and cytokines produced by HTLV-1-infected T cells impair the ability of astrocytes to manage the steady-state level of glutamate, which in turn may affect neuronal and oligodendrocytic functions and survival.