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Mauro Maurantonio

Researcher at University of Modena and Reggio Emilia

Publications -  18
Citations -  657

Mauro Maurantonio is an academic researcher from University of Modena and Reggio Emilia. The author has contributed to research in topics: Nonalcoholic fatty liver disease & Diabetes mellitus. The author has an hindex of 11, co-authored 18 publications receiving 521 citations.

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Nonalcoholic fatty liver disease: Evolving paradigms

TL;DR: NAFLD is highly prevalent in certain cohorts of individuals who are thus potentially amenable to selective screening strategies, intensive follow-up schedules for early identification of liver-related and extrahepatic complications and in whom earlier and more aggressive treatment schedules should be carried out whenever possible.
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Cardiovascular risk, lipidemic phenotype and steatosis. A comparative analysis of cirrhotic and non-cirrhotic liver disease due to varying etiology.

TL;DR: CVR is increased in ALD, NAFLD and chronic HCV infection, all conditions featuring IR and steatosis, and irrespective of serum lipid phenotype, hepatic ste atosis and IR may be major shared determinants in amplifying CVR in common liver disease due to varying etiology.
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Gender-differences in disease distribution and outcome in hospitalized elderly: data from the REPOSI study.

Salvatore Corrao, +313 more
TL;DR: The study showed a gender dimorphism in the demographic and morbidity profiles of hospitalized elderly people, emphasizing once more the need for a personalized process of healthcare.
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Adherence to antithrombotic therapy guidelines improves mortality among elderly patients with atrial fibrillation: insights from the REPOSI study

Marco Proietti, +332 more
TL;DR: Non-adherence to guidelines is highly prevalent among elderly AF patients, despite guideline-adherent treatment being independently associated with lower risk of all-cause and CV deaths.
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A round trip from nonalcoholic fatty liver disease to diabetes: molecular targets to the rescue?

TL;DR: T2D and NAFLD are mutually, closely andBi-directionally associated and an improved understanding of molecular pathogenesis underlying this bi-directional association may allow us to be able to prevent the development of T2D by halting the progression ofNAFLD.