M
Michael A. Long
Researcher at New York University
Publications - 51
Citations - 4597
Michael A. Long is an academic researcher from New York University. The author has contributed to research in topics: Zebra finch & Biological neural network. The author has an hindex of 24, co-authored 45 publications receiving 3993 citations. Previous affiliations of Michael A. Long include University of Texas at Austin & McGovern Institute for Brain Research.
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Journal ArticleDOI
Electrical synapses in the mammalian brain.
Barry W. Connors,Michael A. Long +1 more
TL;DR: Electrical synapses are a ubiquitous yet underappreciated feature of neural circuits in the mammalian brain and may be electrically coupled by other connexin types or by pannexins, a newly described family of gap junction proteins.
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Using temperature to analyse temporal dynamics in the songbird motor pathway
Michael A. Long,Michale S. Fee +1 more
TL;DR: Local manipulation of brain temperature should be broadly applicable to the identification of neural circuitry that controls the timing of behavioural sequences and, more generally, to the study of the origin and role of oscillatory and other forms of brain dynamics in neural systems.
Journal ArticleDOI
Electrical synapses in the thalamic reticular nucleus
Carole E. Landisman,Michael A. Long,Michael A. Long,Michael Beierlein,Michael Beierlein,Michael R. Deans,David L. Paul,David L. Paul,Barry W. Connors,Barry W. Connors +9 more
TL;DR: It is concluded that Cx36-dependent gap junctions play an important role in the regulation of neural firing patterns within the TRN, and electrical synapses are a common mechanism for generating synchrony within networks of inhibitory neurons in the mammalian forebrain.
Journal ArticleDOI
Support for a synaptic chain model of neuronal sequence generation
TL;DR: This paper found that the subthreshold membrane potential is characterized by a large rapid depolarization 5-10 ms prior to burst onset, consistent with a synaptically-connected chain of neurons in HVC.
Journal ArticleDOI
A viral strategy for targeting and manipulating interneurons across vertebrate species
Jordane Dimidschstein,Jordane Dimidschstein,Qian Chen,Robin Tremblay,Stephanie L. Rogers,Giuseppe A. Saldi,Lihua Guo,Lihua Guo,Qing Xu,Qing Xu,Runpeng Liu,Congyi Lu,Jianhua Chu,Joshua S. Grimley,Anne-Rachel F. Krostag,Ajamete Kaykas,Michael C. Avery,Mohammad S. Rashid,Myungin Baek,Amanda L. Jacob,Gordon B. Smith,Daniel E. Wilson,Georg Kosche,Illya Kruglikov,Tomasz Rusielewicz,Vibhakar C. Kotak,Todd M. Mowery,Stewart A. Anderson,Edward M. Callaway,Jeremy S. Dasen,David Fitzpatrick,Valentina Fossati,Michael A. Long,Scott Noggle,John V. Reynolds,Dan H. Sanes,Bernardo Rudy,Guoping Feng,Guoping Feng,Gord Fishell,Gord Fishell +40 more
TL;DR: In this article, a recombinant adeno-associated virus that restricts gene expression to GABAergic interneurons within the telencephalon was proposed to study brain function in both mice and non-genetically tractable species.