M
Miia Kivipelto
Researcher at Karolinska University Hospital
Publications - 516
Citations - 70761
Miia Kivipelto is an academic researcher from Karolinska University Hospital. The author has contributed to research in topics: Dementia & Population. The author has an hindex of 91, co-authored 447 publications receiving 58328 citations. Previous affiliations of Miia Kivipelto include National Institute for Health and Welfare & National Institutes of Health.
Papers
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Sleep disturbances and dementia risk: A multicenter study
Shireen Sindi,Shireen Sindi,Ingemar Kåreholt,Ingemar Kåreholt,Lena Johansson,Johan Skoog,Linnea Sjöberg,Hui-Xin Wang,Hui-Xin Wang,Boo Johansson,Laura Fratiglioni,Hilkka Soininen,Alina Solomon,Alina Solomon,Ingmar Skoog,Miia Kivipelto +15 more
TL;DR: Few longitudinal studies assessed whether sleep disturbances are associated with dementia risk, but research is needed to establish a clear association between sleep disturbances and dementia risk.
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Cortical thickness in frontotemporal dementia, mild cognitive impairment, and Alzheimer's disease
Päivi Hartikainen,Janne V. Räsänen,Valtteri Julkunen,Eini Niskanen,Merja Hallikainen,Miia Kivipelto,Ritva Vanninen,Anne M. Remes,Hilkka Soininen +8 more
TL;DR: The patterns of regions of thinning in FTD when compared to controls and also S-MCI patients showed similar trends; thinning of the bilateral frontal poles and bilateral medial temporal lobe structures, especially the anterior part of the gingulum, the uncus, and parahippocampal gyri.
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APOE-ε4 is associated with weight loss in women with AD A population-based study
Matti Vanhanen,Miia Kivipelto,Keijo Koivisto,Johanna Kuusisto,Leena Mykkänen,Eeva-Liisa Helkala,T. Hänninen,K. Kervinen,Y. A. Kesäniemi,Mikko P. Laakso,H. Soininen,Markku Laakso +11 more
TL;DR: The APOE-ε4 allele may contribute to the unexplained weight loss in AD, especially in women, when men and women were analyzed separately, and weight loss was observed only in those women with AD with the ε4 allele.
Commentary Cholinesterase inhibition: is there evidence for disease-modifying effects?
TL;DR: It is plausible that cholinesterase inhibition might contribute to disease modification, and the detection of putative disease-modifying effects may be most easily implemented in certain patient subpopulations, and genotyping studies suggest a particular role for BuChE.
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Whole brain atrophy rate predicts progression from MCI to Alzheimer's disease.
Gabriela Spulber,Eini Niskanen,Stuart W. S. MacDonald,Oded Smilovici,Kewei Chen,Eric M. Reiman,Anne M. Jauhiainen,Merja Hallikainen,Susanna Tervo,Lars-Olof Wahlund,Ritva Vanninen,Miia Kivipelto,Hilkka Soininen +12 more
TL;DR: This study demonstrates an association between whole brain atrophy rate and subsequent rate of clinical progression from MCI to AD, and suggests that IPCA could be an effective brain-imaging marker of progression to AD and useful tool for the evaluation of disease-modifying treatments.