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Mohammed R. Milad

Researcher at New York University

Publications -  140
Citations -  17366

Mohammed R. Milad is an academic researcher from New York University. The author has contributed to research in topics: Extinction (psychology) & Fear conditioning. The author has an hindex of 52, co-authored 135 publications receiving 15054 citations. Previous affiliations of Mohammed R. Milad include University of Illinois at Chicago & Nathan Kline Institute for Psychiatric Research.

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Neurons in medial prefrontal cortex signal memory for fear extinction

TL;DR: It is suggested that consolidation of extinction learning potentiates infralimbic activity, which inhibits fear during subsequent encounters with fear stimuli, indicating that medial prefrontal cortex might store long-term extinction memory.
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Biological studies of post-traumatic stress disorder

TL;DR: This Review attempts to present the current state of understanding of post-traumatic stress disorder on the basis of psychophysiological, structural and functional neuroimaging, and endocrinological, genetic and molecular biological studies in humans and in animal models.
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Neurobiological basis of failure to recall extinction memory in posttraumatic stress disorder.

TL;DR: Findings support the hypothesis that fear extinction is impaired in PTSD and suggest that dysfunctional activation in brain structures that mediate fear extinction learning, and especially its recall, underlie this impairment.
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Fear Extinction as a Model for Translational Neuroscience: Ten Years of Progress

TL;DR: Research in fear extinction could serve as a model for translational research in other areas of behavioral neuroscience, and new approaches to understanding and exploiting fear extinction are highlighted.
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Recall of fear extinction in humans activates the ventromedial prefrontal cortex and hippocampus in concert.

TL;DR: Results support the involvement of the human hippocampus as well as vmPFC in the recall of extinction memory and provide a paradigm for future investigations of fronto-temporal function during extinction recall in psychiatric disorders such as posttraumatic stress disorder.