M
Moritz Pfeiffenberger
Researcher at Humboldt University of Berlin
Publications - 22
Citations - 272
Moritz Pfeiffenberger is an academic researcher from Humboldt University of Berlin. The author has contributed to research in topics: Bone healing & Mesenchymal stem cell. The author has an hindex of 6, co-authored 18 publications receiving 138 citations. Previous affiliations of Moritz Pfeiffenberger include Leibniz Association & Charité.
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Journal ArticleDOI
Journey into Bone Models: A Review.
Julia Scheinpflug,Moritz Pfeiffenberger,Alexandra Damerau,Alexandra Damerau,Franziska Schwarz,Martin Textor,Annemarie Lang,Annemarie Lang,Frank Schulze +8 more
TL;DR: The current review summarizes and discusses the latest developments in bone tissue engineering and organoid culture including suitable cell sources, extracellular matrices and microfluidic bioreactor systems.
Journal ArticleDOI
Antibodies against chemokine receptors CXCR3 and CXCR4 predict progressive deterioration of lung function in patients with systemic sclerosis
Florian Weigold,Jeannine Günther,Moritz Pfeiffenberger,Otavio Cabral-Marques,Elise Siegert,Duska Dragun,Aurélie Philippe,Ann-Katrin Regensburger,Andreas Recke,Xinhua Yu,Frank Petersen,Rusan Catar,Robert Biesen,Falk Hiepe,Gerd R Burmester,Harald Heidecke,Gabriela Riemekasten +16 more
TL;DR: Anti-CXCR3/4 abs and their corresponding receptors are linked with the severity of SSc-ILD and could be used for risk stratification.
Journal ArticleDOI
Collagen I-based scaffolds negatively impact fracture healing in a mouse-osteotomy-model although used routinely in research and clinical application.
Annemarie Lang,Marieluise Kirchner,Jonathan Stefanowski,Mattea Durst,Marie-Christin Weber,Moritz Pfeiffenberger,Alexandra Damerau,Anja E. Hauser,Paula Hoff,Georg N. Duda,Frank Buttgereit,Katharina Schmidt-Bleek,Timo Gaber,Timo Gaber +13 more
TL;DR: The results of this study show for the first time a negative influence of both ACS-H and ACS-L on bone formation demonstrating a substantial need for more sophisticated delivery systems for local stimulation of bone healing in both clinical application and research.
Journal ArticleDOI
CTLA-4 Mediates Inhibitory Function of Mesenchymal Stem/Stromal Cells
Timo Gaber,Kerstin Schönbeck,Kerstin Schönbeck,Holger Hoff,Cam Loan Tran,Cam Loan Tran,Cindy Strehl,Cindy Strehl,Annemarie Lang,Annemarie Lang,Sarah Ohrndorf,Moritz Pfeiffenberger,Moritz Pfeiffenberger,Eric Röhner,Georg Matziolis,Gerd-R. Burmester,Gerd-R. Burmester,Frank Buttgereit,Frank Buttgereit,Paula Hoff +19 more
TL;DR: It is shown that depending on the environmental conditions, MSCs express different isoforms of CTLA-4 with the secreted isoform (sCTLA- 4) being the most abundant under hypoxic conditions and the immunosuppressive function of M SCs is mediated mainly by the secretion of CTla-4.
Journal ArticleDOI
Impact of Janus Kinase Inhibition with Tofacitinib on Fundamental Processes of Bone Healing.
Timo Gaber,Timo Gaber,Antonia Clara Katharina Brinkman,Antonia Clara Katharina Brinkman,Justyna Pienczikowski,Justyna Pienczikowski,Karoline Diesing,Karoline Diesing,Alexandra Damerau,Alexandra Damerau,Moritz Pfeiffenberger,Moritz Pfeiffenberger,Annemarie Lang,Annemarie Lang,Sarah Ohrndorf,Gerd-Rüdiger Burmester,Gerd-Rüdiger Burmester,Frank Buttgereit,Frank Buttgereit,Paula Hoff,Paula Hoff +20 more
TL;DR: The data indicate that tofacitinib may influence bone healing by promotion of hMSC recruitment into the hypoxic microenvironment of the fracture gap but does not interfere with the cartilaginous phase of the soft callus phase of fracture healing process, which could in part explain the positive impact of tofacITinib on bone erosions in RA.