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Myriam M. Chaumeil

Researcher at University of California, San Francisco

Publications -  49
Citations -  2003

Myriam M. Chaumeil is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Isocitrate dehydrogenase & Mutant. The author has an hindex of 25, co-authored 44 publications receiving 1709 citations. Previous affiliations of Myriam M. Chaumeil include University of California, Berkeley & Temple University.

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Gene Expression Profile Identifies Tyrosine Kinase c-Met as a Targetable Mediator of Antiangiogenic Therapy Resistance

TL;DR: Findings support the role of c-Met in survival in hypoxia and invasion, features associated with antiangiogenic therapy resistance, and growth and therapeutic resistance of xenografts resistant to antiangIogenic therapy.
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Noninvasive detection of target modulation following phosphatidylinositol-3-kinase inhibition using hyperpolarized 13C magnetic resonance spectroscopy

TL;DR: The application of hyperpolarized (13)C MRS of pyruvate to monitor alterations in LDHA activity and expression caused by PI3K pathway inhibition is illustrated, showing the potential of this method for noninvasive imaging of drug target modulation.
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Lactate dehydrogenase A silencing in IDH mutant gliomas.

TL;DR: It is demonstrated that LDHA was silenced in IDH(mt) derived BTSCs, including those that did not retain the mutant IDH1 allele (mIDH(wt), matched BTSC xenografts, and parental glioma tissues, and this is the first demonstration of downregulation of LDHA in cancer.
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Non-invasive in vivo assessment of IDH1 mutational status in glioma

TL;DR: It is shown that [1-(13)C] α-ketoglutarate can serve as a metabolic imaging agent for non-invasive, real-time, in vivo monitoring of mutant IDH1 activity, and can inform onIDH1 status.
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IDH1 Mutation Induces Reprogramming of Pyruvate Metabolism

TL;DR: It is established that the IDH1 mutation induces an MRS-detectable reprogramming of pyruvate metabolism, which is essential for cell proliferation and clonogenicity, with immediate therapeutic implications.