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Nathalie Pizzinat
Researcher at French Institute of Health and Medical Research
Publications - 28
Citations - 1116
Nathalie Pizzinat is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Monoamine oxidase & Monoamine neurotransmitter. The author has an hindex of 16, co-authored 27 publications receiving 939 citations. Previous affiliations of Nathalie Pizzinat include Paul Sabatier University & University of Toulouse.
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Journal ArticleDOI
CD4+ T Cells Promote the Transition From Hypertrophy to Heart Failure During Chronic Pressure Overload
Fanny Laroumanie,Victorine Douin-Echinard,Joffrey Pozzo,Olivier Lairez,Florence Tortosa,Claire Vinel,Christine Delage,Denis Calise,Marianne Dutaur,Angelo Parini,Nathalie Pizzinat +10 more
TL;DR: A crucial role is demonstrated for the first time a crucial role of CD4+ T cells and specific antigen recognition in the progression from compensated cardiac hypertrophy to HF.
Journal ArticleDOI
The elusive family of imidazoline binding sites
TL;DR: Angelo Parini and colleagues summarize the evidence for the characterization and location of imidazoline binding sites, and speculate on the clinical implications of compounds acting on these sites.
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Regulation of JNK/ERK activation, cell apoptosis, and tissue regeneration by monoamine oxidases after renal ischemia-reperfusion.
Oxana Kunduzova,Pascale Bianchi,Nathalie Pizzinat,Ghislaine Escourrou,Marie-Hélène Seguelas,Angelo Parini,Claudie Cambon +6 more
TL;DR: The crucial role of MAOs in mediating the production of injurious ROS, which contribute to acute apoptotic and necrotic cell death induced by renal ischemia‐reperfusion in vivo, is demonstrated.
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Reactive oxygen species production by monoamine oxidases in intact cells.
TL;DR: Results show that, in a normal cell environment, MAO can be a source of reactive oxygen species which could have a functional impact on cell functions and is proposed as a rapid and sensitive procedure to characterize the monoamine oxidase isoforms and their regulation in intact cells.
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Platelet derived serotonin drives the activation of rat cardiac fibroblasts by 5-HT2A receptors
Samiye Yabanoglu,Mansour Akkiki,Mansour Akkiki,Marie-Hélène Seguelas,Marie-Hélène Seguelas,Jeanne Mialet-Perez,Jeanne Mialet-Perez,Angelo Parini,Angelo Parini,Nathalie Pizzinat,Nathalie Pizzinat +10 more
TL;DR: It is demonstrated for the first time that factors released from platelet directly regulate cardiac fibroblasts by enhancing secretion of TGF-beta1 and MMPs and promoting their migration and differentiation.