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Nattawat Onlamoon

Researcher at Mahidol University

Publications -  57
Citations -  2400

Nattawat Onlamoon is an academic researcher from Mahidol University. The author has contributed to research in topics: Dengue virus & Dengue fever. The author has an hindex of 20, co-authored 56 publications receiving 2115 citations. Previous affiliations of Nattawat Onlamoon include Yerkes National Primate Research Center & Emory University.

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Human antibody responses after dengue virus infection are highly cross-reactive to Zika virus

TL;DR: It is shown that both acute and convalescent dengue sera potently bind and neutralize ZIKV and that this cross-reactivity is also evident at the monoclonal level, suggesting that preexisting immunity to DENV may impact protective immune responses against ZikV.
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Rapid and massive virus specific plasmablast responses during acute dengue virus infection in humans

TL;DR: Very potent plasmablast responses that often increased more than 1,000-fold over the baseline levels in healthy volunteers are found, raising the question as to whether these cells might have a role in dengue immunopathology during the ongoing infection.
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Dengue Virus Infection Induces Expansion of a CD14+CD16+ Monocyte Population that Stimulates Plasmablast Differentiation

TL;DR: A systems biological approach to analyze immune responses to dengue in humans revealed that genes encoding proinflammatory mediators and type I interferon-related proteins were associated with high DENV levels during initial symptomatic disease and CD14(+)CD16(+) monocytes increased in the blood.
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Autoimmune Mechanisms as the Basis for Human Peripartum Cardiomyopathy

TL;DR: Findings for the first time suggest that such abnormalities may in concert lead to the initiation and perpetuation of an autoimmune process, which leads to cardiac failure and disease.
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Dengue virus–induced hemorrhage in a nonhuman primate model

TL;DR: Rhesus macaques inoculated intravenously with a high dose of d Dengue virus produced dengue hemorrhage, which may provide a unique platform to define the early events in dengingue virus infection and help identify which blood components contribute to the pathogenesis of denge disease.