Nikolaj H.T. Petersen

TL;DR: Yeast two‐hybrid screening revealed that MKS1 interacts with the WRKY transcription factors WRKY25 and WRKY33, and may contribute to MPK4‐regulated defense activation by coupling the kinase to specific WR KY transcription factors.

TL;DR: Autophagic cell death contributes to HR PCD and can function in parallel with other prodeath pathways, and it is demonstrated that PCD triggered by coiled-coil (CC)-type immune receptors via NDR1 is either autophagy-independent or engages autophagic components with cathepsins and other unidentified cell death mediators.

TL;DR: It is suggested that no single spinal mechanism is responsible for the development of spasticity but that failure of different spinal inhibitory mechanisms (reciprocal IA inhibition, presynaptic inhibition, IB inhibition, recurrent inhibition) are involved in different patients depending on the site of lesion and the etiology of the spastic symptoms.

TL;DR: The Arabidopsis ACD11 gene encodes a sphingosine transfer protein and was identified by the accelerated cell death phenotype of the loss of function acd11 mutant, which exhibits heightened expression of genes involved in the disease resistance hypersensitive response (HR).

TL;DR: It is demonstrated that transgenic expression in Arabidopsis thaliana of human GLTP partially suppressed the phenotype of the acd11 null mutant, resulting in delayed programmed cell death development and plant survival, and Surprisingly, a GLTP mutant form impaired in glycolipid transfer activity also complemented the acD11 mutants.