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Nima Hemmat

Researcher at Tabriz University of Medical Sciences

Publications -  45
Citations -  901

Nima Hemmat is an academic researcher from Tabriz University of Medical Sciences. The author has contributed to research in topics: Cancer & Medicine. The author has an hindex of 10, co-authored 35 publications receiving 339 citations.

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A Systematic Review on the Therapeutic Potentiality of PD-L1-Inhibiting MicroRNAs for Triple-Negative Breast Cancer: Toward Single-Cell Sequencing-Guided Biomimetic Delivery.

TL;DR: In this paper, the authors conducted a systematic review based on the preferred reporting items for systematic reviews and meta-analyses (PRISMA) to provide a comprehensive and unbiased synthesis of currently available evidence regarding the effect of PD-L1-inhibiting miRs restoration on TNBC development and tumor microenvironment.
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Immune checkpoints in tumor microenvironment and their relevance to the development of cancer stem cells

TL;DR: The CSCs immunological mechanisms are summarized and interactions between these cells and factors that are present in the TME to repress immune system responses and enhance tumor survival are discussed.
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The Positive and Negative Immunoregulatory Role of B7 Family: Promising Novel Targets in Gastric Cancer Treatment.

TL;DR: In this paper, the authors present eleven known members of the B7 family as immune checkpoint molecules: B7-1 (CD80), B72 (CD86), C7-H1 (PD-L1, CD274), B 7-DC (PDCD1LG2, PD-L2, CD273), C 7-H2 (B7RP1, ICOS-L, CD275), C 3-H3 (CD276), C 4-H5 (VISTA, Gi24, DD1α, Dies1
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Arginase 1 ( Arg1 ) as an Up-Regulated Gene in COVID-19 Patients: A Promising Marker in COVID-19 Immunopathy.

TL;DR: In this paper, the qPCR results showed that the expression of Arg1 was significantly increased in COVID-19 patients compared to healthy individuals (p < 0.0002 and AUC = 0.8401).
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Viral infection and atherosclerosis.

TL;DR: This review considers the available data about the effects and correlations of DNA and RNA viruses on atherosclerosis and the role of viral infections in atherogenesis.