P
Paolo Pelosi
Researcher at University of Genoa
Publications - 975
Citations - 47463
Paolo Pelosi is an academic researcher from University of Genoa. The author has contributed to research in topics: Mechanical ventilation & Lung injury. The author has an hindex of 93, co-authored 852 publications receiving 37918 citations. Previous affiliations of Paolo Pelosi include University of Insubria & Heidelberg University.
Papers
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Journal Article
Predictive scores for postoperative pulmonary complications: time to move towards clinical practice.
Lorenzo Ball,Paolo Pelosi +1 more
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Intra-abdominal hypertension, prone ventilation, and abdominal suspension.
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Fluids in ARDS: more pros than cons
Renata de S. Mendes,Paolo Pelosi,Marcus J. Schultz,Marcus J. Schultz,Marcus J. Schultz,Patricia R. M. Rocco,Pedro L. Silva +6 more
TL;DR: In acute respiratory distress syndrome (ARDS), increased pulmonary vascular permeability makes the lung vulnerable to edema, and the use of conservative as compared to liberal fluid strategies may increase the number of ventilator-free days and survival, as well as reduce organ dysfunction as mentioned in this paper.
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Ligand-binding properties of three odorant-binding proteins of the diamondback moth Plutella xylostella
TL;DR: This work measured the affinities of three selected OBPs to a series of potential odorants and found one compound is particularly interesting being part of the chemical pathway leading to regeneration of lignin, one of the defense strategies of the plant against insect attack and might find applications as a repellent for P. xylostella and other pests.
Journal ArticleDOI
The impact of fluid status and decremental PEEP strategy on cardiac function and lung and kidney damage in mild-moderate experimental acute respiratory distress syndrome.
Nazareth N. Rocha,Cynthia S. Samary,Mariana A. Antunes,Milena V. Oliveira,Matheus R. Hemerly,Patrine S. Santos,Vera Luiza Capelozzi,Fernanda F. Cruz,John J. Marini,Pedro L. Silva,Paolo Pelosi,Patricia R. M. Rocco +11 more
TL;DR: In this article, the effects of abrupt versus gradual PEEP decrease, combined with standard versus high volume fluid administration, on cardiac function, as well as lung and kidney damage in an established model of mild-moderate acute respiratory distress syndrome (ARDS).