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Peter Pruisscher

Researcher at Stockholm University

Publications -  10
Citations -  300

Peter Pruisscher is an academic researcher from Stockholm University. The author has contributed to research in topics: Diapause & Population. The author has an hindex of 6, co-authored 8 publications receiving 153 citations.

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Unprecedented reorganization of holocentric chromosomes provides insights into the enigma of lepidopteran chromosome evolution.

TL;DR: This work investigates an unprecedented reorganization of the standard lepidopteran chromosome structure in the green-veined white butterfly (Pieris napi) and finds that gene content in P. napi has been extensively rearranged in large collinear blocks, suggesting both a mechanism and a possible role for selection in determining the boundaries of these genome-wide rearrangements.
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Genetic variation underlying local adaptation of diapause induction along a cline in a butterfly.

TL;DR: In this paper, the authors investigate the genetic variation underlying latitudinal variation in diapause induction and the selection dynamics acting upon it, using a genomewide scan for divergent regions between two populations of the butterfly Pararge aegeria that differ strongly in their induction thresholds.
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Effect of winter cold duration on spring phenology of the orange tip butterfly, Anthocharis cardamines

TL;DR: The overall post‐winter pupal development time, following removal from winter cold, was negatively related to cold duration, through a combined effect of cold duration on diapause duration and on post‐diapause development time.
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Energy and lipid metabolism during direct and diapause development in a pierid butterfly.

TL;DR: The data indicate that the diapause lipidomic phenotype is developed early, when pupae are still at high temperature, and retained until post-diapause development, indicating that once diAPause is terminated, development proceeds at a low rate even at low temperature.
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Sex-linked inheritance of diapause induction in the butterfly Pieris napi

TL;DR: The results indicate a strongly heritable diapause induction with a major component on the Z‐chromosome, as well as a minor effect of the autosomal background.