P
Peter W. Reeh
Researcher at University of Erlangen-Nuremberg
Publications - 187
Citations - 13190
Peter W. Reeh is an academic researcher from University of Erlangen-Nuremberg. The author has contributed to research in topics: Calcitonin gene-related peptide & TRPV1. The author has an hindex of 61, co-authored 180 publications receiving 12118 citations. Previous affiliations of Peter W. Reeh include Heidelberg University & University of Pécs.
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Journal ArticleDOI
Modulation of CGRP and PGE2 release from isolated rat skin by alpha-adrenoceptors and kappa-opioid-receptors.
TL;DR: It is concluded that NE affected stimulated PGE2 release via alpha2-adrenoceptors on cells other than cutaneous afferents while kappa-opioid receptors are expressed in peripheral terminals of cutaneousAfferents and their activation reduced CGRP release and secondary P GE2 formation.
Journal ArticleDOI
Effects of classical algogens
Peter W. Reeh,Michaela Kress +1 more
TL;DR: Recruitment of unmyelinated afferents and sustained non-adapting activity occur when moderately low pH and inflammatory mediators combine to mimick the inflammatory exclude, which maintains a primary afferent input from inflamed tissue to the central nervous system.
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Crotalphine desensitizes TRPA1 ion channels to alleviate inflammatory hyperalgesia.
Elisangela Bressan,Filip Touska,Irina Vetter,Katrin Kistner,Tatjana I. Kichko,Nathalia B. Teixeira,Gisele Picolo,Yara Cury,Richard J. Lewis,Michael Fischer,Katharina Zimmermann,Peter W. Reeh +11 more
TL;DR: Results suggest that crotalphine is the first peptide to mediate antinociception selectively and at subnanomolar concentrations by targeting TRPA1 ion channels.
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Responsiveness of C-fiber nociceptors to punctate force-controlled stimuli in isolated rat skin: lack of modulation by inflammatory mediators and flurbiprofen
TL;DR: Even refined mechanostimulation was unable to demonstrate sensitization of the predominant nociceptor classes in the rat skin, and low-in contrast to higher-threshold units showed greater dynamic sensitivity and response magnitude.
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Pulmonary serotonin 5-HT3-sensitive afferent fibers modulate renal sympathetic nerve activity in rats.
Roland Veelken,M. Leonard,Alexander Stetter,Karl F. Hilgers,Johannes F.E. Mann,Peter W. Reeh,Helmut Geiger,Friedrich C. Luft +7 more
TL;DR: The decreased RSNA in the absence of cardiac and abdominal vagal afferents and the strong response of 20% of pulmonary single fibers to intravenous PBG suggest that pulmonary fibers play a role in a 5-HT3 serotenergic reflex, suggesting pulmonary serotonin could influence the neural control of renal function.