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Roberta Venturella

Researcher at Magna Græcia University

Publications -  111
Citations -  2612

Roberta Venturella is an academic researcher from Magna Græcia University. The author has contributed to research in topics: Uterine fibroids & Ovarian reserve. The author has an hindex of 23, co-authored 94 publications receiving 1718 citations.

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Journal ArticleDOI

Psychological impact of coronavirus disease 2019 in pregnant women.

Gabriele Saccone, +9 more
- 07 May 2020 - 
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Follicular versus luteal phase ovarian stimulation during the same menstrual cycle (DuoStim) in a reduced ovarian reserve population results in a similar euploid blastocyst formation rate: new insight in ovarian reserve exploitation.

Filippo Maria Ubaldi, +9 more
- 01 Jun 2016 - 
TL;DR: Stimulation with an identical protocol in the FP and LP of the same menstrual cycle resulted in a similar number of blastocysts in patients with reduced ovarian response, thus increasing the number of patients undergoing transfer per menstrual cycle.
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Prophylactic salpingectomy in premenopausal low-risk women for ovarian cancer: Primum non nocere

Michele Morelli, +7 more
- 01 Jun 2013 - 
TL;DR: The addition of bilateral salpingectomy to TLH for prevention of ovarian cancer in women who do not carry a BRCA1/2 mutations do not show negative effects on the ovarian function and no perioperative complications are related to the salpedectomy step in TLH.
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Risk factors associated with adverse fetal outcomes in pregnancies affected by Coronavirus disease 2019 (COVID-19): a secondary analysis of the WAPM study on COVID-19

Daniele Di Mascio, +205 more
- 26 Nov 2020 - 
TL;DR: Early gestational age at infection, maternal ventilatory supports and low birthweight are the main determinants of adverse perinatal outcomes in fetuses with maternal COVID-19 infection.
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Understanding Ovarian Hypo-Response to Exogenous Gonadotropin in Ovarian Stimulation and Its New Proposed Marker-The Follicle-To-Oocyte (FOI) Index.

Carlo Alviggi, +8 more
- 17 Oct 2018 - 
TL;DR: The role of FOI is contextualized as a parameter to identify this condition, the underlying mechanisms potentially implicated in the pathogenesis of hypo-response are discussed, and possible treatment strategies to overcome hyper-responsiveness to gonadotropin stimulation are appraised.