S
Sabine Sarnacki
Researcher at Necker-Enfants Malades Hospital
Publications - 214
Citations - 6682
Sabine Sarnacki is an academic researcher from Necker-Enfants Malades Hospital. The author has contributed to research in topics: Medicine & Transplantation. The author has an hindex of 38, co-authored 188 publications receiving 5599 citations. Previous affiliations of Sabine Sarnacki include French Institute of Health and Medical Research & Paris Descartes University.
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Journal ArticleDOI
Monitoring of blood vessels and tissues by a population of monocytes with patrolling behavior.
Cédric Auffray,Darin K. Fogg,Meriem Garfa,Gaelle Elain,Olivier Join-Lambert,Olivier Join-Lambert,Samer Kayal,Samer Kayal,Samer Kayal,Sabine Sarnacki,Sabine Sarnacki,Ana Cumano,Grégoire Lauvau,Frederic Geissmann,Frederic Geissmann,Frederic Geissmann +15 more
TL;DR: It is shown, by direct examination of blood monocyte functions in vivo, that a subset of monocytes patrols healthy tissues through long-range crawling on the resting endothelium, which initiated an early immune response and differentiated into macrophages.
Journal ArticleDOI
Guidelines for Imaging and Staging of Neuroblastic Tumors: Consensus Report from the International Neuroblastoma Risk Group Project
Hervé Brisse,M. Beth McCarville,Claudio Granata,K. Barbara Krug,Sandra L. Wootton-Gorges,Kimio Kanegawa,Francesco Giammarile,Matthias Schmidt,Barry L. Shulkin,Katherine K. Matthay,Katherine K. Matthay,Val Lewington,Sabine Sarnacki,Barbara Hero,Michio Kaneko,Wendy B. London,Andrew D.J. Pearson,Susan L. Cohn,Tom Monclair +18 more
TL;DR: The present consensus report was written by the INRG Imaging Committee to optimize imaging and staging and reduce interobserver variability and contribute substantially to more uniform staging and thereby facilitate comparisons of clinical trials conducted in different parts of the world.
Journal ArticleDOI
Targeted therapy in patients with PIK3CA-related overgrowth syndrome
Quitterie Venot,Thomas Blanc,Thomas Blanc,Thomas Blanc,Smail Hadj Rabia,Smail Hadj Rabia,Laureline Berteloot,Sophia Ladraa,J P Duong,J P Duong,Estelle Blanc,Simon C. Johnson,Clément Hoguin,Olivia Boccara,Sabine Sarnacki,Sabine Sarnacki,Nathalie Boddaert,Nathalie Boddaert,Stéphanie Pannier,Stéphanie Pannier,Frank Martinez,Sato Magassa,Junna Yamaguchi,Bertrand Knebelmann,Bertrand Knebelmann,Bertrand Knebelmann,Pierre Merville,Nicolas Grenier,Dominique Joly,Dominique Joly,Dominique Joly,Valérie Cormier-Daire,Valérie Cormier-Daire,Caroline Michot,Caroline Michot,Christine Bole-Feysot,Arnaud Picard,Arnaud Picard,Véronique Soupre,Stanislas Lyonnet,Stanislas Lyonnet,Jérémy Sadoine,Lotfi Slimani,Catherine Chaussain,Cécile Laroche-Raynaud,Laurent Guibaud,Christine Broissand,Jeanne Amiel,Jeanne Amiel,Christophe Legendre,Christophe Legendre,Christophe Legendre,Fabiola Terzi,Fabiola Terzi,Guillaume Canaud,Guillaume Canaud,Guillaume Canaud +56 more
TL;DR: This study provides the first direct evidence supporting PIK3CA inhibition as a promising therapeutic strategy in patients with PROS, by reversing symptoms in a mouse model of PROS/CLOVES syndrome, which results from gain-of-function mutations in PI3KCA.
Journal ArticleDOI
Germline mutations of the RET ligand GDNF are not sufficient to cause Hirschsprung disease.
Rémi Salomon,T Attié,Anna Pelet,Christelle Bidaud,Charis Eng,Jeanne Amiel,Sabine Sarnacki,Olivier Goulet,Claude Ricour,Claire Nihoul-Fékété,Arnold Munnich,Stanislas Lyonnet +11 more
TL;DR: S segregation analysis suggested an incompletely penetrant dominant inheritance in HSCR families with agan-glionosis extending beyond the sigmoid colon, and mutations of the gene encoding GDNF could either cause or modulate the H SCR phenotype in some cases.
Journal ArticleDOI
Comprehensive Study of the Intestinal Stage of Listeriosis in a Rat Ligated Ileal Loop System
Bénédicte Pron,Claire Boumaila,Francis Jaubert,Sabine Sarnacki,Jean-Paul Monnet,Patrick Berche,Jean-Louis Gaillard +6 more
TL;DR: It is suggested that Peyer’s patches are preferential sites for replication rather than for entry of L. monocytogenes, due to the presence of highly permissive mononuclear cells whose nature remains to be defined.