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Sharmila Majumdar

Researcher at University of California, San Francisco

Publications -  505
Citations -  29773

Sharmila Majumdar is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Osteoarthritis & Cartilage. The author has an hindex of 88, co-authored 477 publications receiving 27074 citations. Previous affiliations of Sharmila Majumdar include University of California & Georgia Regents University.

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Trabecular bone is more deteriorated in spinal cord injured versus estrogen-free postmenopausal women

TL;DR: The effect of unloading on bone architecture is greater than that of estrogen loss in middle-aged women, and postmenopausal, ambulatory women did not show the deteriorated trabeculae that were evident in women with SCI.
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Automatic Hip Fracture Identification and Functional Subclassification with Deep Learning.

TL;DR: A deep learning model identified and classified hip fractures with expert-level performance, at the very least, and when used as an aid, improved human performance, with aided resident performance approximating that of unaided fellowship-trained attending physicians.
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Association of cartilage defects, and other MRI findings with pain and function in individuals with mild–moderate radiographic hip osteoarthritis and controls

TL;DR: Acetabular cartilage defects, but not femoral cartilage defect or ROA, were associated with greater self-reported pain and disability and BMELs and subchondral cysts were related to greater hip related self- reported pain and Disability.
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Hyaluronan increases RANKL expression in bone marrow stromal cells through CD44.

TL;DR: HA activates CD44 to stimulate RANKL expression in bone marrow stromal cells, and cells from CD44−/− mice exhibit thicker cortical bone and a smaller medullary cavity, but indices of bone resorption are not affected.
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A quantitative study of relaxation rate enhancement produced by iron oxide particles in polyacrylamide gels and tissue

TL;DR: The results suggest that several different mechanisms contribute to relaxation effects in the presence of iron oxide particles and they depend strongly on the way the particles are sequestered.