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Stefano Parmigiani

Researcher at University of Parma

Publications -  157
Citations -  8883

Stefano Parmigiani is an academic researcher from University of Parma. The author has contributed to research in topics: House mice & Aggression. The author has an hindex of 46, co-authored 151 publications receiving 8296 citations.

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A Physiologically Based Approach To the Study of Bisphenol a and Other Estrogenic Chemicals On the Size of Reproductive Organs, Daily Sperm Production, and Behavior:

TL;DR: Two chemicals previously shown to have estrogenic activity, bisphenol A and octylphenol, were examined for their effects on accessory reproductive organs and daily sperm production in male offspring of mice fed these chemicals during pregnancy and it was found that the 2 ng/g dose permanently increased the size of the preputial glands, but reduced thesize of the epididymides.
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Prostate enlargement in mice due to fetal exposure to low doses of estradiol or diethylstilbestrol and opposite effects at high doses

TL;DR: It is suggested that a small increase in estrogen may modulate the action of androgen in regulating prostate differentiation, resulting in a permanent increase in prostatic androgen receptors and prostate size.
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Escalated aggressive behavior: dopamine, serotonin and GABA.

TL;DR: This review emphasizes research on escalated forms of aggression in animals and humans and their pharmacotherapy, and points to 5-HT(1B) receptor subtypes as particularly relevant and significant targets for anti-aggressive interventions.
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Why Public Health Agencies Cannot Depend on Good Laboratory Practices as a Criterion for Selecting Data: The Case of Bisphenol A

TL;DR: Differences between industry-funded GLP studies of BPA conducted by commercial laboratories for regulatory purposes and non-GLP studies conducted in academic and government laboratories are reviewed to identify hazards and molecular mechanisms mediating adverse effects.
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Exposure to a low dose of bisphenol A during fetal life or in adulthood alters maternal behavior in mice.

TL;DR: The changes seen in maternal behavior may be the result of a direct effect of BPA on the neuroendocrine substrates underlying the initiation of maternal behavior.