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Stuart M. Cobbe

Researcher at University of Glasgow

Publications -  215
Citations -  31173

Stuart M. Cobbe is an academic researcher from University of Glasgow. The author has contributed to research in topics: Myocardial infarction & Heart failure. The author has an hindex of 64, co-authored 214 publications receiving 30218 citations. Previous affiliations of Stuart M. Cobbe include British Heart Foundation & Glasgow Royal Infirmary.

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Adenosine increases potassium conductance in isolated rabbit atrioventricular nodal myocytes

TL;DR: Adenosine acts at A1 adenosine receptors to suppress spontaneous activity, hyperpolarise membrane potential and induce a time-independent potassium current in AV nodal cells.
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Do radionuclide and echocardiographic techniques give a universal cut off value for left ventricular ejection fraction that can be used to select patients for treatment with ACE inhibitors after myocardial infarction

TL;DR: Measurement of ejection fraction is highly dependent on the method used and it is therefore impossible to quote a universally applicable figure below which an ACE inhibitor should be used after myocardial infarction.
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Effect of statin therapy on muscle symptoms: an individual participant data meta-analysis of large-scale, randomised, double-blind trials

Christina Reith, +127 more
- 01 Aug 2022 - 
TL;DR: An individual participant data meta-analysis of all recorded adverse muscle events in large, long-term, randomised, double-blind trials of statin therapy found a small, clinically insignificant increase in median creatine kinase values of approximately 0·02 times the upper limit of normal.
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Day-to-day variations in inducibility of ventricular tachyarrhythmias during the late postmyocardial infarction phase in conscious dogs.

TL;DR: Variation in the inducibility of sustained ventricular arrhythmias may conceivably complicate the use of results of programmed ventricular stimulation as determinants of risk of sudden death after myocardial infarction.
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Effect of sustained stretch on dispersion of ventricular fibrillation intervals in normal rabbit hearts

TL;DR: Acute ventricular dilatation produces spatially heterogeneous changes in refractoriness which would predispose to the maintenance of reentrant arrhythmias and regional heterogeneity in the areas of greatest reduction in VF interval, which varied between hearts.