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Susan J. Muller

Researcher at University of California, Berkeley

Publications -  88
Citations -  3619

Susan J. Muller is an academic researcher from University of California, Berkeley. The author has contributed to research in topics: Reynolds number & Newtonian fluid. The author has an hindex of 34, co-authored 87 publications receiving 3282 citations. Previous affiliations of Susan J. Muller include Lawrence Berkeley National Laboratory & Center for Advanced Materials.

Papers
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Roles of epithelial cell-derived periostin in TGF-β activation, collagen production, and collagen gel elasticity in asthma

TL;DR: Epithelial cell-derived periostin in asthma has roles in TGF-β activation and collagen gel elasticity in asthma and alters collagen fibrillogenesis or cross-linking and leads to stiffening of the matrix.
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An interlaboratory comparison of measurements from filament-stretching rheometers using common test fluids

TL;DR: In this paper, a filament-stretching extensional rheometer at Monash University and similar rheometers have been designed and built in other laboratories are compared to help validate the basic technique, a collaborative program was undertaken to compare results from several instruments.
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Wettability of silicone-hydrogel contact lenses in the presence of tear-film components

TL;DR: The captive-bubble technique provides contact angles that are relevant to on-eye lens wear and minimizes or even eliminates protein adsorption in both advancing and receding contact angles, which are important to lens wettability performance.
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Microfluidic four-roll mill for all flow types

TL;DR: In this paper, a microfluidic four-roll mill device that can cover the entire spectrum of flow types including purely rotational flow was designed using pseudo-three-dimensional simulations.
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Ex Vivo Sputum Analysis Reveals Impairment of Protease-dependent Mucus Degradation by Plasma Proteins in Acute Asthma

TL;DR: In airway mucus collected from patients with asthma at various time points during acute asthma exacerbation, protease-driven mucus degradation was inhibited at the height of exacerbation but was restored during recovery, identifying a novel mechanism whereby plasma exudation may impair airways mucus clearance.