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Tao Chen

Researcher at Fourth Military Medical University

Publications -  128
Citations -  11519

Tao Chen is an academic researcher from Fourth Military Medical University. The author has contributed to research in topics: Long-term potentiation & Excitatory postsynaptic potential. The author has an hindex of 36, co-authored 111 publications receiving 8564 citations. Previous affiliations of Tao Chen include Xi'an Jiaotong University & UPRRP College of Natural Sciences.

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Interference with KCTD9 inhibits NK cell activation and ameliorates fulminant liver failure in mice

TL;DR: It is proposed that target inhibition of KCTD9 may prohibit NK cells activity and thus ameliorate liver damage in viral fulminant hepatitis therapy, mediated by impairment of NK cell activation.
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Microglial polarization in TBI: Signaling pathways and influencing pharmaceuticals

TL;DR: This study reviews signaling pathways involved in microglial polarization and medications that influence this process and identifies several drugs that target these pathways, including fingolimod, minocycline, Tak-242 and erythropoietin (EPO), and CSF-1.
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CD4-CD8-T cells contribute to the persistence of viral hepatitis by striking a delicate balance in immune modulation

TL;DR: This study identifiedDN T cells contributing to viral persistence in MHV-3 induced hepatitis in C3H/HeJ mice, which provides a rationale for modulating DN T cells for the management of viral hepatitis.
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Controlled decompression attenuates brain damage in a rat model of epidural extreme intracranial hypertension: Partially via inhibiting necroptosis and inflammatory response

TL;DR: In this paper , a rat model of extreme intracranial hypertension (EEIH) and verified the effectiveness of a surgery method called controlled decompression on attenuating brain injury induced by EEIH.
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Endomorphin-2 Inhibits the Activity of the Spinoparabrachial Projection Neuron through Presynaptic Mechanisms in the Spinal Dorsal Horn in Rats.

TL;DR: Applying EM2 to the SDH could produce analgesia through inhibiting the activities of the spinoparabrachial PNs in lamina I by reducing presynaptic neurotransmitters release from the primary afferent terminals.