T
Thomas Christensen
Researcher at University of Copenhagen
Publications - 145
Citations - 3285
Thomas Christensen is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Stroke & Medicine. The author has an hindex of 28, co-authored 132 publications receiving 2948 citations. Previous affiliations of Thomas Christensen include Aarhus University & Copenhagen University Hospital.
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Journal ArticleDOI
Self-monitoring of oral anticoagulation: systematic review and meta-analysis of individual patient data
Carl Heneghan,Alison Ward,Rafael Perera,Clare Bankhead,Alice Fuller,Richard Stevens,Kairen Bradford,S Tyndel,Pablo Alonso-Coello,Jack Ansell,Jack Ansell,Rebecca J. Beyth,Artur Bernardo,Thomas Christensen,M. E. Cromheecke,Robert Edson,David Fitzmaurice,Alain Gadisseur,Josep M. García-Alamino,Chris Gardiner,Michael Hasenkam,Alan K. Jacobson,Scott Kaatz,Farhad Kamali,Tayyaba Khan,Eve Knight,Heinrich Körtke,Marcel Levi,David B. Matchar,David B. Matchar,David B. Matchar,Bárbara Menéndez-Jándula,Ivo Rakovac,Christian Schaefer,Andrea Siebenhofer,Juan Carlos Souto,Rubina Sunderji,Rubina Sunderji,Kenneth Gin,Kenneth Gin,Karen F. Shalansky,Karen F. Shalansky,Heinz Völler,Otto Wagner,Armin Zittermann +44 more
TL;DR: The analysis showed that self-monitoring and self-management of oral coagulation is a safe option for suitable patients of all ages and patients should also be offered the option to self-manage their disease with suitable health-care support as back-up.
Journal ArticleDOI
Ischemic Damage in Hippocampal CA1 is Dependent on Glutamate Release and Intact Innervation from CA3
Helene Benveniste,Martin Balslev Jørgensen,Mats Sandberg,Thomas Christensen,Henrik Hagberg,N. H. Diemer +5 more
TL;DR: The ischemia-induced damage of pyramidal cells in CA1 is dependent on glutamate release and intact innervation from CA3, and the pathogenetic significance of glutamate is investigated.
Journal ArticleDOI
Microglia and macrophages are major sources of locally produced transforming growth factor‐β1 after transient middle cerebral artery occlusion in rats
Elin Lehrmann,Reinhard Kiefer,Thomas Christensen,Klaus V. Toyka,Jens Zimmer,Nils Henrik Diemer,Hans-Peter Hartung,Bente Finsen +7 more
TL;DR: Activated microglia and macrophages are established as the major source of TGF‐β1 mRNA following experimental focal cerebral ischemia, and TGF•β1‐mediated functions may be exerted bymicroglia both in the early degenerative phase, and later in combination with blood‐borne macrophage, in the remodeling and healing phase after focal cerebral waschemia.
Journal ArticleDOI
Microglial and macrophage reactions mark progressive changes and define the penumbra in the rat neocortex and striatum after transient middle cerebral artery occlusion.
TL;DR: The present results are interpreted as delineating two differentially progressing penumbral zones, which are likely to reflect differences in the underlying degenerative processes.
Journal ArticleDOI
miRNA Expression Profiles in Cerebrospinal Fluid and Blood of Patients with Acute Ischemic Stroke
Sofie Sølvsten Sørensen,Ann-Britt Nygaard,Ming-Yuan Nielsen,Kai Arne Jensen,Thomas Christensen +4 more
TL;DR: The preliminary results demonstrate that it is possible to detect and profile miRNAs in CSF and blood from patients with neurological diseases.