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Thomas M. Badger

Researcher at University of Arkansas for Medical Sciences

Publications -  305
Citations -  13313

Thomas M. Badger is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Soy protein & Offspring. The author has an hindex of 63, co-authored 299 publications receiving 12304 citations. Previous affiliations of Thomas M. Badger include University of Arkansas & United States Department of Agriculture.

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Journal ArticleDOI

Chronic ethanol consumption leads to disruption of vitamin D3 homeostasis associated with induction of renal 1,25 dihydroxyvitamin D3-24-hydroxylase (CYP24A1).

Kartik Shankar, +6 more
- 01 Apr 2008 - 
TL;DR: The data suggest that EtOH reduces circulating 1,25 (OH)2 D3 concentrations as the result of CYP24A1 induction that is mediated via MAPK activation resulting from renal oxidative stress produced by local metabolism of EtOH via CYP2E1 and antidiuretic hormone-1.
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Estradiol Protects against Ethanol-Induced Bone Loss by Inhibiting Up-Regulation of Receptor Activator of Nuclear Factor-κB Ligand in Osteoblasts

Jin-Ran Chen, +8 more
- 01 Dec 2006 - 
TL;DR: E2 prevents EtOH-induced bone loss by opposing the induction of RANKL mRNA in osteoblasts and ethanol-induced osteoclastogenesis, through opposing effects on sustained ERK signaling.
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Long-Term Administration of Gonadotropin-Releasing Hormone in Men with Idiopathic Hypogonadotropic Hypogonadism: A Model for Studies of the Hormone's Physiologic Effects

Daniel I. Spratt, +6 more
- 01 Dec 1986 - 
TL;DR: Men with idiopathic hypogonadotropic hypog onadotropin-releasing hormone (GnRH) present a useful model to study the onset and maintenance of reproductive function in men.
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Immunohistochemical characterization of hepatic malondialdehyde and 4-hydroxynonenal modified proteins during early stages of ethanol-induced liver injury.

Brante P. Sampey, +4 more
- 01 Jun 2003 - 
TL;DR: An involvement of both MDA and 4-HNE in the process of liver fibrosis is supported by the observation that these aldehydic products of lipid peroxidation increase procollagen type I messenger RNA and protein in cultured human stellate cells.
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Physiologic and genomic analyses of nutrition-ethanol interactions during gestation: Implications for fetal ethanol toxicity.

Kartik Shankar, +7 more
- 01 Sep 2006 - 
TL;DR: The data suggest that undernutrition potentiates the fetal toxicity of EtOH in part by disrupting maternal GH-IGF-1, signaling thereby decreasing maternal uterine capacity and placental growth.